p53 inhibits asynchronous neurotransmission mediated by VAMP4, 13/October/2013, 23.09

Idea Proposed by: Dr L Boominathan Ph.D. Terms & Conditions apply http://genomediscovery.org/registration/terms-and-conditions/ To cite: Boominathan, p53 inhibits asynchronous neurotransmission mediated by VAMP4, 13/October/2013, 23.09, Genome-2-Bio-Medicine Discovery center (GBMD), http://genomediscovery.org * Research cooperation Netradicinė medicina, sveikata, dietos, sveika mityba netradicinemedicina.com

p73 inhibits metaphase-to-anaphase transition in mouse oocytes via down regulation of its target gene, 13/October/2013, 23.06

Idea Proposed by: Dr L Boominathan Ph.D. Terms & Conditions apply http://genomediscovery.org/registration/terms-and-conditions/ To cite: Boominathan, p73 inhibits metaphase-to-anaphase transition in mouse oocytes via down regulation of its target gene, 13/October/2013, 23.06, Genome-2-Bio-Medicine Discovery center (GBMD), http://genomediscovery.org * Research cooperation

Connecting with Nobel’s (Sudhof C Thomas) favorite work: p53/p63/p73 inhibits IGF-1 exocytosis via up regulation of its target miRNAs, 13/October/2013, 22.28

This year’s Nobel prize winner for Physiology or Medicine Prof. Sudhof C Thomas (from Stanford University, California, USA) has published a paper in the Journal Cell (2011) stating that ” Activity-dependent IGF-1 exocytosis is controlled by the Calcium-sensor synaptotoagmin-10.” This study suggests that deletion of synaptotoagmin-10 results in (i) impaired secretion of IGF-1; (ii)  smaller neurons; and (iii) an overall decrease in synapse […]

Connecting with Nobel's (Sudhof C Thomas) favorite work: p53/p63/p73 inhibits IGF-1 exocytosis via up regulation of its target miRNAs, 13/October/2013, 22.28

This year’s Nobel prize winner for Physiology or Medicine Prof. Sudhof C Thomas (from Stanford University, California, USA) has published a paper in the Journal Cell (2011) stating that ” Activity-dependent IGF-1 exocytosis is controlled by the Calcium-sensor synaptotoagmin-10.” This study suggests that deletion of synaptotoagmin-10 results in (i) impaired secretion of IGF-1; (ii)  smaller neurons; and (iii) an overall decrease in synapse […]