Natural product-based therapy for T2DM: Kirenol, a natural compound isolated from Siegesbeckiae, inhibits gluconeogenesis and hyperglycemia via up regulation of Cyclin D1, 25/February/2014, 23.23

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A study from the Department of Cancer Biology, Dana-Farber Cancer Institute;  Department of Cell Biology, Harvard Medical School, Boston, Massachusetts, USA; and others shows that Cyclin D1–Cdk4 controls glucose metabolism independently of cell cycle progression.”

This study was published in the June 26, 2014 Nature [I.F >42] by Prof. Puigserver and others from the Department of Cancer Biology, Dana-Farber Cancer Institute;  Department of Cell Biology, Harvard Medical School, Boston, Massachusetts, USA.

On the foundation of this interesting finding, Dr L Boominathan, Director-cum-chief Scientist of GBMDreports here that: Natural productbased therapy for T2DM: Kirenol, a natural compound isolated from Siegesbeckiae, inhibits gluconeogenesis and hyperglycemia via up regulation of Cyclin D1. This study suggests that Kirenol by up regulating its target gene CyclinD1, it may inhibit  gluconeogenesis and hyperglycemia. Together, this study suggests that pharmacological formulations encompassing “Kirenol or its analogues  can be used in the treatment of NIDDM.

Idea Proposed/Formulated byDr L Boominathan Ph.D.

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To citeBoominathan, L., Natural productbased therapy for T2DM: Kirenol, a natural compound isolated from Siegesbeckiae, inhibits gluconeogenesis and hyperglycemia via up regulation of Cyclin D1, 25/February/2014, 23.23, Genome-2-Bio-Medicine Discovery center (GBMD), http://genomediscovery.org

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