Turning off inflammation inhibits autoimmune myocarditis: ATRA/Tretinoin, A derivative of Vitamin-A, inhibits cardiac inflammation and injury via down regulation of its target gene, 18/March/2015, 14.00

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A recent study from the Department of Cardiology and Pneumology, Charité University Medicine Berlin, Campus Benjamin Franklin, Hindenburgdamm 30, Berlin 12200, Germany shows that “Adiponectin protects against Toll-like receptor 4-mediated cardiac inflammation and injury.” This study was published in the 1 August’13 issue of the Journal “Cardiovascular Research” by Prof. Skurk C., Dr. Jenke A and others.

On the foundation of this interesting finding, Dr L Boominathan PhD, Director-cum-chief Scientist of GBMD, reports that: Turning off inflammation inhibits autoimmune myocarditis: ATRA/Tretinoin, A derivative of Vitamin-A, inhibits cardiac inflammation and injury via down regulation of its target gene. Thus, pharmacological formulations encompassing Tretinoin/ATRA or its analogues” may be used to treat patients suffering from autoimmune myocarditis.

Idea Proposed/Formulated byDr L Boominathan Ph.D.

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To citeBoominathan,  Turning off inflammation inhibits autoimmune myocarditis: ATRA/Tretinoin, A derivative of Vitamin-A, inhibits cardiac inflammation and injury via down regulation of its target gene, 18/March/2015,  14.00,  Genome-2-Bio-Medicine Discovery center (GBMD), http://genomediscovery.org

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* Research cooperation

Undisclosed information: How ATRA/Tretinoin suppresses cardiac inflammation and injury

* Research cooperation

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