Molecular therapy for Myocardial Infarction: Ars2 (arsenite-resistance protein 2/Serrate RNA effector molecule homolog) improves ageing- and stress-associated decline in cardiac function via up regulation of PNUTS/PPP1R10 (Serine/threonine-protein phosphatase 1 regulatory subunit 10), 5/April/2015, 14.33

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A recent study from the Institute for Cardiovascular RegenerationCentre of Molecular Medicine, Frankfurt, Germany shows that “MicroRNA-34a regulates cardiac ageing and function.” This study was published in the 7 March  2013 issue of Nature  by Prof Dimmler, Boon, and others.

On the foundation of this interesting finding, Dr L Boominathan PhD, Director-cum-chief Scientist of GBMD, reports that: Molecular therapy for Myocardial Infarction: Ars2 (arsenite-resistance protein 2/Serrate RNA effector molecule homolog) improves ageing- and stress-associated decline in cardiac function via up regulation of PNUTS/PPP1R10 (Serine/threonine-protein phosphatase 1 regulatory subunit 10). By treating aged people, who are susceptible to cardiac diseases, with Ars2 activators, one may prevent ageing-associated decline in cardiac function. Together, this study suggests, for the first time, that pharmacological formulations encompassing “Ars2 activators” may be used to improve cardiac function following myocardial infarction.

Idea Proposed/Formulated byDr L Boominathan Ph.D.

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Undisclosed information: How Ars2 increases the expression of PNUTS/PPP1R10

To citeBoominathan, Molecular therapy for Myocardial Infarction: Ars2 (arsenite-resistance protein 2/Serrate RNA effector molecule homolog) improves ageing- and stress-associated decline in cardiac function via up regulation of PNUTS/PPP1R10 (Serine/threonine-protein phosphatase 1 regulatory subunit 10), 5/April/2015, 14.33,  Genome-2-Bio-Medicine Discovery center (GBMD), http://genomediscovery.org

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