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Dr.Boomi

Natural product-based Life-extension therapy: Epigallocatechin-3-Gallate, the major component of Green tea, increases life span by inducing the expression of its target gene, 22/February/2015, 11.27 am
Natural product-based Life-extension therapy: Epigallocatechin-3-Gallate, the major component of Green tea, increases life span by inducing the expression of its target gene, 22/February/2015, 11.27 am 150 150

Idea Proposed/Formulated by: Dr L Boominathan Ph.D. Web: http://genomediscovery.org Significance: Epigallocatechin-3-Gallate, the major component of Green tea, may increase life span by inducing its target gene. Together, pharmacological formulations encompassing “Epigallocatechin-3-Gallate or its analogues“ may function as longevity-promoting agents. Terms…

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Molecular based therapy for NIDDM: Jagged1 inhibits gluconeogenesis and hyperglycemia via up regulation of CyclinD1, 22/February/2014, 9.13 am
Molecular based therapy for NIDDM: Jagged1 inhibits gluconeogenesis and hyperglycemia via up regulation of CyclinD1, 22/February/2014, 9.13 am 150 150

A study from the Department of Cancer Biology, Dana-Farber Cancer Institute;  Department of Cell Biology, Harvard Medical School, Boston, Massachusetts, USA; and others shows that “Cyclin D1–Cdk4 controls glucose metabolism independently of cell cycle progression.”…

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MiRNA-based molecular therapy for Right ventricular cardiomyopathy: Repressor-element-1-silencing transcription factor suppresses arrhythmogenic right ventricular cardiomyopathy via down regulation of its target gene, 22/February/2015, 12.09 am
MiRNA-based molecular therapy for Right ventricular cardiomyopathy: Repressor-element-1-silencing transcription factor suppresses arrhythmogenic right ventricular cardiomyopathy via down regulation of its target gene, 22/February/2015, 12.09 am 150 150

A recent study from the Ludwig Institute for Cancer Research Limited, Nuffield Department of Clinical Medicine, University of Oxford, Oxford OX3 7DQ, United Kingdom shows that “iASPP, a previously unidentified regulator of desmosomes,…

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AntagomiR-based theapy for wound repair: AntagomiR-196 promotes epithelial wound repair via up regulation of its target gene, 21/February/2015, 23.43
AntagomiR-based theapy for wound repair: AntagomiR-196 promotes epithelial wound repair via up regulation of its target gene, 21/February/2015, 23.43 150 150

A recent study from the Epithelial Pathobiology and Mucosal Inflammation Research Unit, Department of Pathology and Laboratory Medicine, Emory University School of Medicine, Atlanta, Georgia, USA shows that “Annexin A1–containing extracellular vesicles and…

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Natural product-based therapy for Metastatic tumors: Resveratrol suppresses the expression of PD-1, a suppressor of activation of T-cells, and activates the immune system via down regulation of its target gene, 21/February/2015, 23.20
Natural product-based therapy for Metastatic tumors: Resveratrol suppresses the expression of PD-1, a suppressor of activation of T-cells, and activates the immune system via down regulation of its target gene, 21/February/2015, 23.20 150 150

Idea Proposed/Formulated by: Dr L Boominathan Ph.D. Terms & Conditions apply http://genomediscovery.org/registration/terms-and-conditions/ Citation: Boominathan, Natural product-based therapy for Metastatic tumors: Resveratrol suppresses the expression of PD-1, a suppressor of activation of T-cells, and activates the immune…

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Natural product-based anticancer therapy: Ursolic acid increases the expression of tumor suppressor RECK via down regulation of its target gene, 21/February/2015, 22.52
Natural product-based anticancer therapy: Ursolic acid increases the expression of tumor suppressor RECK via down regulation of its target gene, 21/February/2015, 22.52 150 150

Idea Proposed/Formulated by: Dr L Boominathan Ph.D. Terms & Conditions apply http://genomediscovery.org/registration/terms-and-conditions/ Citation: Boominathan, Natural product-based anticancer therapy: Ursolic acid increases  the expression of tumor suppressor RECK via down regulation of its target gene,…

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