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October 8, 2016

Natural product-based therapy for Idiopathic pulmonary fibrosis: Resveratrol, a longevity promoting small molecule found in grapes and ground nuts, increases TRF1 expression and ameliorates pulmonary fibrosis via upregulation of its target gene, 8/October/2015, 4.24 pm

A study from the Spanish National Cancer Centre, Melchor Fernández Almagro 3, Madrid, Spain“ shows that: “Mice with Pulmonary Fibrosis Driven by Telomere Dysfunction.” This study was published in the 14 July 2015 issue of […]
October 8, 2016

Natural product therapy for Pulmonary Fibrosis: Pear water extract increases Toll-like receptor 4 expression and extracellular matrix glycosaminoglycan Hyaluronan levels, promotes alveolar progenitor cell renewal and repair of lung injury and prevents severe pulmonary fibrosis via down regulation of its target gene, 8/October/2015, 3.46 pm

A study from the Department of Medicine, Cedars-Sinai Medical Center, Los Angeles, California, USA; and Women’s Guild Lung Institute, Cedars-Sinai Medical Center, Los Angeles, California, USA […]
October 7, 2016

Natural product therapy targeting aberrant Lipid synthesis in Human cancers: Litchi (Litchi chinensis) flower extract suppresses the expression of the principal enzyme in lipid synthesis, ACLY, inhibits aberrant lipid production in cancer cells and tumor progression via up regulation of its target gene, 7/October/2016, 11.53 pm

A recent study from Rutgers Cancer Institute of New Jersey, Rutgers University, The State University of New Jersey, New Brunswick, New Jersey, USA shows that “Cullin3–KLHL25 ubiquitin […]
October 7, 2016

Therapeutic insights into PD-1 pathway blockade for Human cancer therapy: Agaricus blazei Murill (AbM) inhibits the expression of co-inhibitory receptor PD-1 on Antigen- Specific T-cells, increases the production of cytokines by tumor-infiltrating lymphocytes (TIL), and draining lymph nodes and augments anti-tumor activity via down-regulation of its target gene, 7/october/2016, 11.23 pm

A recent study from Division of Infectious Diseases, School of Medicine, Johns Hopkins University, Baltimore, MD 21218, United States shows that “TGF-β1-Mediated Smad3 Enhances PD-1 Expression […]
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