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A study from the Department of Metabolism and Aging, The Scripps Research Institute, Florida, USA shows that “MicroRNA-182 regulates amygdala-dependent memory formation.”

This study was published in the January 23, 2013 J Neurosci. by Miller CAGriggs EM and others from the Department of Metabolism and Aging, The Scripps Research Institute, Florida, USA.

On the foundation of this interesting finding, Dr L Boominathan, Director-cum-chief Scientist of GBMD, reports here that: Insights into Stress-induced Memory loss: Stress-induced Adenocorticotrophic hormone (ACTH) disrupts auditory fear memory formation by suppressing the expression of cortactin and Rac1. This study suggests that ACTH, by increasing its target gene, it may suppress the expression of synapse-enriched regulators of actin  such as, Cortactin and Rac1. Together, this study suggests that pharmacological formulations encompassing “ACTH inhibitors”   may be used to treat patients suffering from stress-mediated memory loss.

Idea Proposed/Formulated byDr L Boominathan Ph.D.

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To citeBoominathan, L., Insights into Stress-induced Memory loss: Adenocorticotrophic hormone (ACTH) disrupts auditory fear memory formation by suppressing the expression of Cortactin and Rac1Genome-2-Bio-Medicine Discovery center (GBMD), 19/08/2014, 11.40 am,  http://genomediscovery.org

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