A study from the Institute for Molecular Systems Biology, ETH Zurich, Zurich, Switzerland shows that “MicroRNAs 103 and 107 regulate insulin sensitivity.”
This study was published in the June 8, 2011 Nature [I.F >35] by Prof. Stoffel M, Trajkovski M and others from the he Institute for Molecular Systems Biology, ETH Zurich, Zurich, Switzerland.
On the foundation of this interesting finding, Dr L Boominathan, Director-cum-chief Scientist of GBMD, reports here that: Insights into the treatment of T2D: IGF-1 increases insulin sensitivity via up regulation of a regulator of insulin receptor, Caveolin-1 . This study suggests that IGF-1, by down regulating its target gene, it may up regulate the regulator of insulin receptor, caveolin-1. Together, this study suggests that pharmacological formulations encompassing “IGF-1 or its activators” may be used in the treatment of NIDDM.
Idea Proposed/Formulated by: Dr L Boominathan Ph.D.
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To cite: Boominathan, L., Insights into the treatment of T2D: IGF-1 increases Insulin sensitivity via up regulation of a regulator of insulin receptor, Caveolin-1, Genome-2-Bio-Medicine Discovery center (GBMD), 17/08/2014, 15.00, http://genomediscovery.org
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