A recent study from the Department of Microbiology and Immunobiology, Harvard Medical School, Boston, Massachusetts; Department of Pediatrics, Baylor College of Medicine, Houston, Texas, USA; and Institute for Biomedical Research, University College London, London, UK shows that “Nociceptive sensory neurons drive interleukin-23-mediated psoriasiform skin inflammation.” This study was published in the Apr 23 2014 Nature by Prof Ulrich H. von Andrian, Lorena Riol-Blanco, and others.
On the foundation of this interesting finding, Dr L Boominathan PhD, Director-cum-chief Scientist of GBMD, reports that: Mechanistic and therapeutic insights into the treatment of Psoriasis: Curcumin, the major component of turmeric, inhibits psoriasiform skin inflammation via up regulation of its target gene. This study suggests that Curcumin, by increasing the expression of its target gene, it may alleviate the interleukin-23-mediated psoriasiform skin inflammation. Together, this study suggests that pharmacological formulations encompassing” Curcumin or its analogues” may be used to treat psoriasiform skin inflammation.
Idea Proposed/Formulated by: Dr L Boominathan Ph.D.
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To cite: Boominathan, L., Mechanistic and therapeutic insights into the treatment of Psoriasis: Curcumin, the major component of turmeric, inhibits psoriasiform skin inflammation via up regulation of its target gene, 29/July/2014, 17.39, Genome-2-Bio-Medicine Discovery center (GBMD), http://genomediscovery.org
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