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This year’s Nobel prize winner for Physiology or Medicine Prof. Sudhof (Stanford University, California, USA) has published a paper in Nature Neuroscience (2009) stating that “Synaptotagmin-1 functions as a calcium sensor for spontaneous release.” 

In connection with this finding, Dr Boominathan, Founder Director-cum-chief scientist of GBMD, reports that The tumor suppressor p73 functions as a calcium sensor for spontaneous release via Synaptoagmin-1. This finding suggests that the expression of p63/p73/p53 may regulate spontaneous Neurotransmitter release via Synaptoagmin-1 Based on this result Dr Boominathan believes that p53/p63/p73 may play a critical role in the disease pathogenic mechanisms of diabetics.

Idea Proposed byDr L Boominathan Ph.D.

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To citeBoominathan, p73 connects with Nobel Laureate’s(Sudhof) finding: The tumor suppressor p73 functions as a calcium sensor for spontaneous Neurotransmitter release via Synaptoagmin-1, 9/October/2013, 5.37 am,  Genome-2-Bio-Medicine Discovery center (GBMD), http://genomediscovery.org

* Research cooperation

 


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