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A study from the Department of Biochemistry-Molecular Biology, Peking University Health Science Center, Beijing 100191, China has reported that “Tumor suppressor p53 cooperates with SIRT6 to regulate gluconeogenesis by promoting FoxO1 nuclear exclusion.”

This study was published in the July 09, 2014 Proc Natl Acad Sci U S A.  [I.F >10] by Prof. Zhu WG, Zhang and others from  the Department of Biochemistry-Molecular Biology, Peking University Health Science Center, Beijing 100191, China.

On the foundation of this interesting finding, Dr L Boominathan, Director-cum-chief Scientist of GBMD, reports here that: Physiological mechanisms that Control Gluconeogensis: The stem cell and reprogramming protein Oct-4 increases the expression of phosphoenolpyruvate carboxykinase (PCK1) via up regulation of its target gene.  

Idea Proposed/Formulated by: Dr L Boominathan PhD

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CitationBoominathan, Physiological mechanisms that Control Gluconeogensis: The stem cell and reprogramming protein Oct-4 increases the expression of phosphoenolpyruvate carboxykinase (PCK1) via up regulation of its target gene, 23/July/2014, 6.06 am,  Genome-2-Bio-Medicine Discovery center (GBMD), http://genomediscovery.org

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