Yoga therapy for Alzheimer’s disease: Nisshesha rechaka pranayama (Brief Intermittent Hypoxia) protects against amyloid deposition and toxicity in Alzheimer’s disease via up regulation of its target gene, 6/October/2014, 7.09 am

Yoga therapy for Alzheimer’s disease: Nisshesha rechaka pranayama (Brief Intermittent Hypoxia) protects against amyloid deposition and toxicity in Alzheimer’s disease via up regulation of its target gene, 6/October/2014, 7.09 am

Yoga therapy for Alzheimer’s disease: Nisshesha rechaka pranayama (Brief Intermittent Hypoxia) protects against amyloid deposition and toxicity in Alzheimer’s disease via up regulation of its target gene, 6/October/2014, 7.09 am 150 150 Dr Boomi's Genom-2-Discovery Center

A recent study from the Gladstone Institute of Neurological Disease, San Francisco, California, USA shows that Progranulin protects against amyloid β deposition and toxicity in Alzheimer’s disease mouse models. This study was published in the 28 September 2014 Nature  by Prof Gan Li, S Sakura Minami, and others.

On the foundation of this interesting finding, Dr L Boominathan PhD, Director-cum-chief Scientist of GBMD, reports that: Yoga therapy for Alzheimer’s disease: Nisshesha rechaka pranayama (Brief Intermittent Hypoxia) protects against amyloid deposition and toxicity in Alzheimer’s disease via up regulation of its target gene. Nisshesha rechaka pranayama  (Brief Intermittent Hypoxia), by increasing the expression of its target gene in Neurons, it may protect against amyloid deposition and toxicity. Thereby, it may inhibit the progression of Alzheimer’s disease; and  prevent ageing-associated decline in brain function. Together, this study suggests that breathing exercise (PranayamaBrief Intermittent Hypoxiamay function as an adjuvant therapy for patients suffering from Alzheimer’s disease.

Idea Proposed/Formulated byDr L Boominathan Ph.D.

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To citeBoominathan, Yoga therapy for Alzheimer’s disease: Nisshesha rechaka pranayama (Brief Intermittent Hypoxia) protects against amyloid deposition and toxicity in Alzheimer’s disease via up regulation of its target gene, 6/October/2014,  7.09 am,  Genome-2-Bio-Medicine Discovery center (GBMD), http://genomediscovery.org

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