Ageing

Natural product-based regenerative therapy for improving cognitive deficits in aged individuals: Sulforaphane, isolated from Broccoli and others, decreases vascular cell adhesion molecule 1 (VCAM 1), inhibits microglia activity, increases hippocampal neural precursor activity, ameliorates cognition, & attenuates age-related neurodegeneration, through up regulation of its target genes, 14/June/2019, 6.31 am
Natural product-based regenerative therapy for improving cognitive deficits in aged individuals: Sulforaphane, isolated from Broccoli and others, decreases vascular cell adhesion molecule 1 (VCAM 1), inhibits microglia activity, increases hippocampal neural precursor activity, ameliorates cognition, & attenuates age-related neurodegeneration, through up regulation of its target genes, 14/June/2019, 6.31 am 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A study from the Department of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, CA, USA, VA Palo Alto Health Care System, Palo Alto,…

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A derivative of male hormone Dihydrotestosterone (DHT)  boosts longevity: Dihydrotestosterone (DHT) slows down cardiac aging, and increases lifespan, via up-regulation of BubR1 and others genes, 17/April/2019, 11.28 pm
A derivative of male hormone Dihydrotestosterone (DHT)  boosts longevity: Dihydrotestosterone (DHT) slows down cardiac aging, and increases lifespan, via up-regulation of BubR1 and others genes, 17/April/2019, 11.28 pm 1280 720 Dr Boomi's Genom-2-Discovery Center

 What they say: Introduction:  A recent study from the Department of Genetics, Paul F. Glenn Laboratories for the Biological Mechanisms of Aging Harvard Medical School, Boston, USA; and Department of Pharmacology,…

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Ribonucleic acid-based therapy for inflammation and arthritis:  LncRNA ZFAS1 (zinc finger antisense (ZFAS)1 increases IL-9 levels, augments proliferation of ILC2s (type 2 innate lymphoid cells) and regulatory T (Treg) cells, promotes resolution of inflammation, attenuates cartilage destruction, decreases bone loss, and inhibits pathological features associated with arthritis via up-regulation of its target genes, 3/March/2018, 9.13 am
Ribonucleic acid-based therapy for inflammation and arthritis:  LncRNA ZFAS1 (zinc finger antisense (ZFAS)1 increases IL-9 levels, augments proliferation of ILC2s (type 2 innate lymphoid cells) and regulatory T (Treg) cells, promotes resolution of inflammation, attenuates cartilage destruction, decreases bone loss, and inhibits pathological features associated with arthritis via up-regulation of its target genes, 3/March/2018, 9.13 am 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A study from Department of Internal Medicine 3-Rheumatology and Immunology, Friedrich Alexander University Erlangen-Nürnberg (FAU) and Universitätsklinikum Erlangen, Erlangen, Germany shows that “Resolution of inflammation by…

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Ribonucleic acid-based therapy for aging-associated diseases and Lifespan extension: LncRNA CCAT1  inhibits the interaction of DBC1 with PARP1, increases the PARP1 activity, promotes DNA repair, augments tolerance against radiation, cancer, and aging, via down-regulation of its target gene, 3/March/2019, 8.50 am
Ribonucleic acid-based therapy for aging-associated diseases and Lifespan extension: LncRNA CCAT1  inhibits the interaction of DBC1 with PARP1, increases the PARP1 activity, promotes DNA repair, augments tolerance against radiation, cancer, and aging, via down-regulation of its target gene, 3/March/2019, 8.50 am 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A study from Department of Genetics, Paul F. Glenn Center for the Biology of Aging, Harvard Medical School, Boston, MA, USA; and Department of Pharmacology, School…

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Ribonucleic acid-based therapy for aging-associated diseases and Lifespan extension: LncRNA Zeb2-AS1 increases NMN/NAD levels, decreases interaction of DBC1 with PARP1, increases PARP1 activity, promotes DNA repair, augments tolerance against radiation, cancer and aging via down-regulation of its target gene, 12/January/2019, 6.39 am
Ribonucleic acid-based therapy for aging-associated diseases and Lifespan extension: LncRNA Zeb2-AS1 increases NMN/NAD levels, decreases interaction of DBC1 with PARP1, increases PARP1 activity, promotes DNA repair, augments tolerance against radiation, cancer and aging via down-regulation of its target gene, 12/January/2019, 6.39 am 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A study from Department of Genetics, Paul F. Glenn Center for the Biology of Aging, Harvard Medical School, Boston, MA, USA; and Department of Pharmacology, School…

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RNA-based therapy for aging-associated diseases and Lifespan extension: LncRNA BRM inhibits the interaction of DBC1 with PARP1, increases the PARP1 activity, promotes DNA repair, augments tolerance against radiation, cancer and aging, via down-regulation of its target gene, 4/January/2019, 10.03 am
RNA-based therapy for aging-associated diseases and Lifespan extension: LncRNA BRM inhibits the interaction of DBC1 with PARP1, increases the PARP1 activity, promotes DNA repair, augments tolerance against radiation, cancer and aging, via down-regulation of its target gene, 4/January/2019, 10.03 am 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A study from Department of Genetics, Paul F. Glenn Center for the Biology of Aging, Harvard Medical School, Boston, MA, USA; and Department of Pharmacology, School…

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