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Welcome to Dr.Boomi's

Genome-2-Bio-Medicine Discovery Centre (GBMD)
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Focused on exploring and publishing insights into how energy drives biological cause-and-effect relationships.

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Welcome to Dr.Boomi's

Shaping Science Through Unique Ideas
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Unraveling the power of energy in shaping the intricate dance of biological cause and effect.

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Dr L. Boominathan Ph.D.

President & Director-cum-Chief scientist

Dr.Boominathan was the first one in the world to propose a p73/p63-dependent cancer suppressor pathway (Supported by a legally valid Invention disclosure document certified by an advocate-2002/3)

Empowering Innovation:

Who We Are and What We Create

We generate bio-medical/pharma/therapeutic/clinical ideas for the scientific community. Good Ideas are uncommon and unique. This is because everyone’s educational background is different. As a result, every one’s thinking process is different. Consequently, his/her ability to connect/understand complex biological/pathological mechanism differs.

Total No of Ideas : 11717

LncRNA-based regenerative cardiovascular therapy:  Sirtuin 5 inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration, and survival after myocardial infarction, via up-regulation of its target gene, 14/December/2021, 6.38 am 960 720
  • Dr.Boomi

LncRNA-based regenerative cardiovascular therapy:  Sirtuin 5 inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration, and survival after myocardial infarction, via up-regulation of its target gene, 14/December/2021, 6.38 am

What they say:  A recent study from the Institute for Cardiovascular Regeneration, Centre of Molecular Medicine, Frankfurt, Germany shows that “MicroRNA-34a regulates cardiac ageing and function.” This study was published, in the 7 March  2013 issue of the journal Nature,  by Profs. Stefanie Dimmeler (Director, Center for Molecular Medicine), Reinier A Boon and others. What we say:  On the foundation of this interesting finding, Dr L Boominathan PhD, Director-cum-chief Scientist…

Molecular therapy for safeguarding your heart against cardiac dysfunction: MiR-488-5p-based therapy for cardiomyocyte proliferation and heart regeneration: MiR-488-5p decreases tumor suppressor Mir-128 and cyclin-dependent kinase inhibitor p27 expression, increases SUZ12 expression, increases Cyclin E and CDK2 expression, promotes proliferation/re-entry of postnatal/adult cardiomyocytes, attenuates fibrosis, ameliorates cardiac dysfunction, and promotes heart repair in response to myocardial infarction, via up-regulation of its target gene, 22/October/2021, 12.07 pm 960 720
  • Dr.Boomi

Molecular therapy for safeguarding your heart against cardiac dysfunction: MiR-488-5p-based therapy for cardiomyocyte proliferation and heart regeneration: MiR-488-5p decreases tumor suppressor Mir-128 and cyclin-dependent kinase inhibitor p27 expression, increases SUZ12 expression, increases Cyclin E and CDK2 expression, promotes proliferation/re-entry of postnatal/adult cardiomyocytes, attenuates fibrosis, ameliorates cardiac dysfunction, and promotes heart repair in response to myocardial infarction, via up-regulation of its target gene, 22/October/2021, 12.07 pm

Introduction: What they say: A recent study from Department of Pathology and Laboratory Medicine, University of Cincinnati College of Medicine, Cincinnati, OH, 45267, USA; Key Laboratory of Molecular Target and Clinical Pharmacology, School of Pharmaceutical Sciences & Fifth Affiliated Hospital, Guangzhou Medical University, Guangzhou, Guangdong, 511436, China; and Department of Pathology and Laboratory Medicine, University…

LncRNA-based regenerative cardiovascular therapy:  LncRNA RMRP inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration, and survival after myocardial infarction, via up-regulation of its target gene, 1/November/2021, 5.23 pm 960 720
  • Dr.Boomi

LncRNA-based regenerative cardiovascular therapy:  LncRNA RMRP inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration, and survival after myocardial infarction, via up-regulation of its target gene, 1/November/2021, 5.23 pm

What they say:  A recent study from the Institute for Cardiovascular Regeneration, Centre of Molecular Medicine, Frankfurt, Germany shows that “MicroRNA-34a regulates cardiac ageing and function.” This study was published, in the 7 March  2013 issue of the journal Nature,  by Profs. Stefanie Dimmeler (Director, Center for Molecular Medicine), Reinier A Boon and others. What we say:  On the foundation of this interesting finding, Dr L Boominathan PhD, Director-cum-chief Scientist…

LncRNA-based regenerative cardiovascular therapy:  LncRNA βFaar inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration, and survival after myocardial infarction, via up-regulation of its target gene, 22/October/2021, 11.33 am 960 720
  • Dr.Boomi

LncRNA-based regenerative cardiovascular therapy:  LncRNA βFaar inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration, and survival after myocardial infarction, via up-regulation of its target gene, 22/October/2021, 11.33 am

What they say:  A recent study from the Institute for Cardiovascular Regeneration, Centre of Molecular Medicine, Frankfurt, Germany shows that “MicroRNA-34a regulates cardiac ageing and function.” This study was published, in the 7 March  2013 issue of the journal Nature,  by Profs. Stefanie Dimmeler (Director, Center for Molecular Medicine), Reinier A Boon and others. What we say:  On the foundation of this interesting finding, Dr L Boominathan PhD, Director-cum-chief Scientist…

MiRNA-488-5p inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration, and survival after myocardial infarction, via up-regulation of its target gene, 14/December/2021, 7.07 am 960 720
  • Dr.Boomi

MiRNA-488-5p inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration, and survival after myocardial infarction, via up-regulation of its target gene, 14/December/2021, 7.07 am

  What they say:  A recent study from the Institute for Cardiovascular Regeneration, Centre of Molecular Medicine, Frankfurt, Germany shows that “MicroRNA-34a regulates cardiac ageing and function.” This study was published, in the 7 March  2013 issue of the journal Nature,  by Profs. Stefanie Dimmeler (Director, Center for Molecular Medicine), Reinier A Boon and others. What we say:  On the foundation of this interesting finding, Dr L Boominathan PhD, Director-cum-chief…

Molecular therapy for safeguarding your heart against cardiac dysfunction: lncRNA βFaar-based therapy for cardiomyocyte proliferation and heart regeneration: lncRNA βFaar decreases tumor suppressor Mir-128 and cyclin-dependent kinase inhibitor p27 expression, increases SUZ12 expression, increases Cyclin E and CDK2 expression, promotes proliferation/re-entry of postnatal/adult cardiomyocytes, attenuates fibrosis, ameliorates cardiac dysfunction, and promotes heart repair in response to myocardial infarction, via up-regulation of its target gene, 22/October/2021, 11.33 am 960 720
  • Dr.Boomi

Molecular therapy for safeguarding your heart against cardiac dysfunction: lncRNA βFaar-based therapy for cardiomyocyte proliferation and heart regeneration: lncRNA βFaar decreases tumor suppressor Mir-128 and cyclin-dependent kinase inhibitor p27 expression, increases SUZ12 expression, increases Cyclin E and CDK2 expression, promotes proliferation/re-entry of postnatal/adult cardiomyocytes, attenuates fibrosis, ameliorates cardiac dysfunction, and promotes heart repair in response to myocardial infarction, via up-regulation of its target gene, 22/October/2021, 11.33 am

Introduction: What they say: A recent study from Department of Pathology and Laboratory Medicine, University of Cincinnati College of Medicine, Cincinnati, OH, 45267, USA; Key Laboratory of Molecular Target and Clinical Pharmacology, School of Pharmaceutical Sciences & Fifth Affiliated Hospital, Guangzhou Medical University, Guangzhou, Guangdong, 511436, China; and Department of Pathology and Laboratory Medicine, University…

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