Diabetes

Molecular therapy for Regenerating the lost pancreatic β-cells in Diabetic patients: ADAR1 (adenosine deaminase acting on RNA increases the expression of IGF1R, INSR, and IRS2, and promotes an insulin-sensitized state, via up-regulation of  its target gene Lin28, 20/December/2018, 10.36 pm
Molecular therapy for Regenerating the lost pancreatic β-cells in Diabetic patients: ADAR1 (adenosine deaminase acting on RNA increases the expression of IGF1R, INSR, and IRS2, and promotes an insulin-sensitized state, via up-regulation of  its target gene Lin28, 20/December/2018, 10.36 pm 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say:   A study from the Stem Cell Transplantation Program, Division of Pediatric Hematology/Oncology, Boston Children’s Hospital and Dana-Farber Cancer Institute, Boston, Massachusetts, USA shows that “The Lin28/let-7 axis regulates glucose metabolism.” This study…

read more

Erythropoietin-based therapy for TIIDM and Metabolic defects: Erythropoietin (EPO)/Hematopoietin increases Sirtuin-4 expression, augments insulin secretion, reduces metabolic stress, improves glucose uptake, promotes glucose homeostasis and prevents progression to TIIDM via down regulation of its target gene, 20/December/2018, 10.28 pm
Erythropoietin-based therapy for TIIDM and Metabolic defects: Erythropoietin (EPO)/Hematopoietin increases Sirtuin-4 expression, augments insulin secretion, reduces metabolic stress, improves glucose uptake, promotes glucose homeostasis and prevents progression to TIIDM via down regulation of its target gene, 20/December/2018, 10.28 pm 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A study from the “Duke Molecular Physiology Institute and Sarah W. Stedman Nutrition and Metabolism Center, Duke University Medical Center, Durham, NC 27701, USA; Department of…

read more

Molecular therapy for TIIDM and Metabolic defects: AP-1 (Activator protein-1) increases Sirtuin-4 expression, augments insulin secretion, reduces metabolic stress, improves glucose uptake, promotes glucose homeostasis and prevents progression to TIIDM via down regulation of its target gene, 20/December/2018, 9.59 pm
Molecular therapy for TIIDM and Metabolic defects: AP-1 (Activator protein-1) increases Sirtuin-4 expression, augments insulin secretion, reduces metabolic stress, improves glucose uptake, promotes glucose homeostasis and prevents progression to TIIDM via down regulation of its target gene, 20/December/2018, 9.59 pm 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A study from the “Duke Molecular Physiology Institute and Sarah W. Stedman Nutrition and Metabolism Center, Duke University Medical Center, Durham, NC 27701, USA; Department of…

read more

Molecular therapy for body weight control, energy homeostasis and TIIDM: Adenosine deaminase acting on RNA 1 (ADAR1) decreases CADM1 and its downstream target genes that inhibit glucose-induced insulin secretion, improves insulin sensitivity, increases energy utilization, promotes weight loss and protects from diet-induced obesity and TIIDM, via up-regulation of its target gene, 20/December/2018, 12.11 pm
Molecular therapy for body weight control, energy homeostasis and TIIDM: Adenosine deaminase acting on RNA 1 (ADAR1) decreases CADM1 and its downstream target genes that inhibit glucose-induced insulin secretion, improves insulin sensitivity, increases energy utilization, promotes weight loss and protects from diet-induced obesity and TIIDM, via up-regulation of its target gene, 20/December/2018, 12.11 pm 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say  A study from the Max Delbrück Center for Molecular Medicine, Berlin, Germany shows that “Regulation of body weight and energy homeostasis by neuronal cell adhesion molecule1.”…

read more

A probiotic way to say no to Diabetes: Probiotic MIYAIRI 588-based therapy for diabetes (TIDM): Probiotic Clostridium butyricum MIYAIRI 588 (CBM588) increases GLP1R and Caveolin-1 (CAV-1) expression, promotes glucose-induced insulin secretion, improves insulin sensitivity, increases energy utilization, promotes weight loss and protects from diet-induced obesity and TIIDM, via up-regulation of its target gene, 17/December/2018, 11.58 pm
A probiotic way to say no to Diabetes: Probiotic MIYAIRI 588-based therapy for diabetes (TIDM): Probiotic Clostridium butyricum MIYAIRI 588 (CBM588) increases GLP1R and Caveolin-1 (CAV-1) expression, promotes glucose-induced insulin secretion, improves insulin sensitivity, increases energy utilization, promotes weight loss and protects from diet-induced obesity and TIIDM, via up-regulation of its target gene, 17/December/2018, 11.58 pm 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A study from the Comprehensive Diabetes Center and Department of Medicine, Division of Endocrinology, Diabetes, and Metabolism, University of Alabama at Birmingham, Birmingham, AL shows that…

read more

A Probiotic way to say no to Diabetes: Probiotic VSL#3-based therapy for diabetes (TIDM): Probiotic VSL#3 increases GLP1R and Caveolin-1 (CAV-1) expression, promotes glucose-induced insulin secretion, improves insulin sensitivity, increases energy utilization, promotes weight loss and protects from diet-induced obesity and TIIDM, via up-regulation of its target gene, 17/December/2018, 12.02 am
A Probiotic way to say no to Diabetes: Probiotic VSL#3-based therapy for diabetes (TIDM): Probiotic VSL#3 increases GLP1R and Caveolin-1 (CAV-1) expression, promotes glucose-induced insulin secretion, improves insulin sensitivity, increases energy utilization, promotes weight loss and protects from diet-induced obesity and TIIDM, via up-regulation of its target gene, 17/December/2018, 12.02 am 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A study from the Comprehensive Diabetes Center and Department of Medicine, Division of Endocrinology, Diabetes, and Metabolism, University of Alabama at Birmingham, Birmingham, AL shows that…

read more
Start Typing