Diabetes

Repurposing the anti-hypertensive agent Aliskiren into an anti-diabetic agent:  Aliskiren (brand name: Tekturna and Rasilez), an antihypertensive agent, increases the expression of IGF1R, INSR, and IRS2, and promotes an insulin-sensitized state, via up-regulation of  its target gene Lin28, 26/October, 2018, 12.15 am
Repurposing the anti-hypertensive agent Aliskiren into an anti-diabetic agent:  Aliskiren (brand name: Tekturna and Rasilez), an antihypertensive agent, increases the expression of IGF1R, INSR, and IRS2, and promotes an insulin-sensitized state, via up-regulation of  its target gene Lin28, 26/October, 2018, 12.15 am 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say:   A study from the Stem Cell Transplantation Program, Division of Pediatric Hematology/Oncology, Boston Children’s Hospital and Dana-Farber Cancer Institute, Boston, Massachusetts, USA shows that “The Lin28/let-7 axis regulates glucose metabolism.” This study…

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A musical way to stay free of diabetes: Music-based therapy for body weight control, energy homeostasis, and TIIDM: Music inhibits DNA methyltransferase 3a (Dnmt3a) expression, increases FGF21 expression, augments insulin sensitivity, increases glucose tolerance, and protects from diet-induced obesity and TIIDM, via up-regulation of its target gene, 22/September/2018, 10.25 pm
A musical way to stay free of diabetes: Music-based therapy for body weight control, energy homeostasis, and TIIDM: Music inhibits DNA methyltransferase 3a (Dnmt3a) expression, increases FGF21 expression, augments insulin sensitivity, increases glucose tolerance, and protects from diet-induced obesity and TIIDM, via up-regulation of its target gene, 22/September/2018, 10.25 pm 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say  A study from Nutritional Sciences and Toxicology Department, University of California, Berkeley, Berkeley, United States shows that “Dnmt3a is an epigenetic mediator of adipose insulin resistance.”…

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Musical way to treat blood sugar disease: Music-based therapy for diabetes (TIIDM): Music increases GLP1R and Caveolin-1 (CAV-1) expression, promotes glucose-induced insulin secretion, improves insulin sensitivity, increases energy utilization, promotes weight loss and protects from diet-induced obesity and TIIDM, via up-regulation of its target gene, 20/October/2018, 5.23 pm
Musical way to treat blood sugar disease: Music-based therapy for diabetes (TIIDM): Music increases GLP1R and Caveolin-1 (CAV-1) expression, promotes glucose-induced insulin secretion, improves insulin sensitivity, increases energy utilization, promotes weight loss and protects from diet-induced obesity and TIIDM, via up-regulation of its target gene, 20/October/2018, 5.23 pm 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A study from the Comprehensive Diabetes Center and Department of Medicine, Division of Endocrinology, Diabetes, and Metabolism, University of Alabama at Birmingham, Birmingham, AL shows that “miR-204 Controls…

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Music as a bio-medicine in the treatment of TIIDM and obesity-associated metabolic deficits: Music increases Lipocalin 2 (LCN2) expression, activates an MC4R-dependent anorexigenic pathway, suppresses appetite and weight gain, increases insulin secretion, improves glucose tolerance, promotes glucose homeostasis, improves obesity-associated metabolic deficits and prevents progression to TIIDM via down-regulation of its target gene, 20/October/2018, 5.08 pm
Music as a bio-medicine in the treatment of TIIDM and obesity-associated metabolic deficits: Music increases Lipocalin 2 (LCN2) expression, activates an MC4R-dependent anorexigenic pathway, suppresses appetite and weight gain, increases insulin secretion, improves glucose tolerance, promotes glucose homeostasis, improves obesity-associated metabolic deficits and prevents progression to TIIDM via down-regulation of its target gene, 20/October/2018, 5.08 pm 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A study from the Department of Physiology-Cellular Biophysics, College of Physicians and Surgeons, Columbia University, New York, New York, USA shows that “MC4R-dependent suppression of appetite by…

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A probiotic way to treat TIIDM and obesity-associated metabolic deficits: Probiotic Lactobaccilus gasseri SBT2055 increases Lipocalin 2 (LCN2) expression, activates an MC4R-dependent anorexigenic pathway, suppresses appetite and weight gain, increases insulin secretion, improves glucose tolerance, promotes glucose homeostasis, improves obesity-associated metabolic deficits and prevents progression to TIIDM via down-regulation of its target gene, 5/October/2018, 11.01 pm
A probiotic way to treat TIIDM and obesity-associated metabolic deficits: Probiotic Lactobaccilus gasseri SBT2055 increases Lipocalin 2 (LCN2) expression, activates an MC4R-dependent anorexigenic pathway, suppresses appetite and weight gain, increases insulin secretion, improves glucose tolerance, promotes glucose homeostasis, improves obesity-associated metabolic deficits and prevents progression to TIIDM via down-regulation of its target gene, 5/October/2018, 11.01 pm 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A study from the Department of Physiology-Cellular Biophysics, College of Physicians and Surgeons, Columbia University, New York, New York, USA shows that “MC4R-dependent suppression of appetite by…

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A probiotic way to stay free of diabetes: Probiotic Lactobacillus gasseri-based therapy for body weight control, energy homeostasis and TIIDM: Probiotic Lactobacillus gasseri inhibits DNA methyltransferase 3a (Dnmt3a) expression, increases FGF21 expression, augments insulin sensitivity, increases glucose tolerance, and protects from diet-induced obesity and TIIDM, via up-regulation of its target gene, 5/September/2018, 10.46 pm
A probiotic way to stay free of diabetes: Probiotic Lactobacillus gasseri-based therapy for body weight control, energy homeostasis and TIIDM: Probiotic Lactobacillus gasseri inhibits DNA methyltransferase 3a (Dnmt3a) expression, increases FGF21 expression, augments insulin sensitivity, increases glucose tolerance, and protects from diet-induced obesity and TIIDM, via up-regulation of its target gene, 5/September/2018, 10.46 pm 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say  A study from Nutritional Sciences and Toxicology Department, University of California, Berkeley, Berkeley, United States shows that “Dnmt3a is an epigenetic mediator of adipose insulin resistance.”…

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