Diabetes

Molecular therapy for Regenerating the lost pancreatic β-cells in Diabetic patients: iASPP increases the expression of IGF1R, INSR, and IRS2, and promotes an insulin-sensitized state via up-regulation of its target gene Lin28, 3/June/2017,11.44 pm
Molecular therapy for Regenerating the lost pancreatic β-cells in Diabetic patients: iASPP increases the expression of IGF1R, INSR, and IRS2, and promotes an insulin-sensitized state via up-regulation of its target gene Lin28, 3/June/2017,11.44 pm 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say:   A study from the Stem Cell Transplantation Program, Division of Pediatric Hematology/Oncology, Boston Children’s Hospital and Dana-Farber Cancer Institute, Boston, Massachusetts, USA shows that “The Lin28/let-7 axis regulates…

read more

Molecular therapy for TIIDM and obesity-associated metabolic deficits: Kallistatin increases Lipocalin 2 (LCN2) expression, activates an MC4R-dependent anorexigenic pathway, suppresses appetite and weight gain, increases insulin secretion, improves glucose tolerance, promotes glucose homeostasis, improves obesity-associated metabolic deficits and prevents progression to TIIDM via down regulation of its target gene, 3/June/2017, 12.15 am
Molecular therapy for TIIDM and obesity-associated metabolic deficits: Kallistatin increases Lipocalin 2 (LCN2) expression, activates an MC4R-dependent anorexigenic pathway, suppresses appetite and weight gain, increases insulin secretion, improves glucose tolerance, promotes glucose homeostasis, improves obesity-associated metabolic deficits and prevents progression to TIIDM via down regulation of its target gene, 3/June/2017, 12.15 am 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A study from the Department of Physiology-Cellular Biophysics, College of Physicians and Surgeons, Columbia University, New York, New York, USA shows that “MC4R-dependent suppression of appetite…

read more

Amino-acid derived regenerative therapy for reversing diabetes: L-Leucine, the branched chain essential amino acid, decreases mTOR expression, promotes regeneration of insulin-producing ß cells, increases insulin secretion, promotes glucose homeostasis and reverses T1D and T2D via down regulation of its target gene, 25/May/2017, 7.21 pm
Amino-acid derived regenerative therapy for reversing diabetes: L-Leucine, the branched chain essential amino acid, decreases mTOR expression, promotes regeneration of insulin-producing ß cells, increases insulin secretion, promotes glucose homeostasis and reverses T1D and T2D via down regulation of its target gene, 25/May/2017, 7.21 pm 150 150 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A study from the Longevity Institute, School of Gerontology, Department of Biological Sciences, University of Southern California, 3715 McClintock Avenue, Los Angeles, California, USA; and Koch Institute…

read more

Molecular therapy for Regenerating the lost pancreatic β-cells in Diabetic patients: GDF11 increases the expression of IGF1R, INSR, and IRS2, and promotes an insulin-sensitized state via up-regulation of its target gene Lin28, 25/May/2017,7.17 am
Molecular therapy for Regenerating the lost pancreatic β-cells in Diabetic patients: GDF11 increases the expression of IGF1R, INSR, and IRS2, and promotes an insulin-sensitized state via up-regulation of its target gene Lin28, 25/May/2017,7.17 am 150 150 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say:   A study from the Stem Cell Transplantation Program, Division of Pediatric Hematology/Oncology, Boston Children’s Hospital and Dana-Farber Cancer Institute, Boston, Massachusetts, USA shows that “The Lin28/let-7 axis regulates…

read more

Natural product-derived (exercise mementics) therapy for Metabolic disease: Angelica sinensis increases PPARδ levels, inhibits glucose catabolism, preserves glucose levels, stimulates FA catabolism, delays the onset of hypoglycemia and boosts endurance exercise via down regulation of its target gene, 19/May/2017, 12.00 am
Natural product-derived (exercise mementics) therapy for Metabolic disease: Angelica sinensis increases PPARδ levels, inhibits glucose catabolism, preserves glucose levels, stimulates FA catabolism, delays the onset of hypoglycemia and boosts endurance exercise via down regulation of its target gene, 19/May/2017, 12.00 am 150 150 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A study from the Gene Expression Laboratory, Salk Institute for Biological Studies, La Jolla, CA 92037, USA; Howard Hughes Medical Institute, Salk Institute for Biological Studies, La Jolla,…

read more
Start Typing