Glaucoma

Hormonal therapy for Glaucoma: Melatonin (N-acetyl-5-methoxy tryptamine), a hormone that regulates sleep and wakefulness, increases Nmnat1 expression, increases NAD+ levels, improves neuronal dysfunction, and protects against glaucoma via down regulation of its target gene, 27/March/2018, 5.41 am

Hormonal therapy for Glaucoma: Melatonin (N-acetyl-5-methoxy tryptamine), a hormone that regulates sleep and wakefulness, increases Nmnat1 expression, increases NAD+ levels, improves neuronal dysfunction, and protects against glaucoma via down regulation of its target gene, 27/March/2018, 5.41 am 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A study from the The Jackson Laboratory, Bar Harbor, ME 04609, USA; Department of Ophthalmology, Tufts University of Medicine, Boston, MA 02111, USA; The Howard Hughes…

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Gaseous therapy for Glaucoma: Hydrogen sulfide, a gaseous neurotransmitter, increases Nmnat1 expression, increases NAD+ levels, improves neuronal dysfunction, and protects against glaucoma via down regulation of its target gene, 26/March/2018, 6.19 am

Gaseous therapy for Glaucoma: Hydrogen sulfide, a gaseous neurotransmitter, increases Nmnat1 expression, increases NAD+ levels, improves neuronal dysfunction, and protects against glaucoma via down regulation of its target gene, 26/March/2018, 6.19 am 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A study from the The Jackson Laboratory, Bar Harbor, ME 04609, USA; Department of Ophthalmology, Tufts University of Medicine, Boston, MA 02111, USA; The Howard Hughes…

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Molecular therapy for Glaucoma: Melatonin (N-acetyl-5-methoxy tryptamine), a regulator of sleep and wakefulness, increases Nmnat1 expression, increases NAD+ levels, improves neuronal dysfunction, and protects against glaucoma via down regulation of its target gene, 10/March/2018, 6.15 am

Molecular therapy for Glaucoma: Melatonin (N-acetyl-5-methoxy tryptamine), a regulator of sleep and wakefulness, increases Nmnat1 expression, increases NAD+ levels, improves neuronal dysfunction, and protects against glaucoma via down regulation of its target gene, 10/March/2018, 6.15 am 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A study from the The Jackson Laboratory, Bar Harbor, ME 04609, USA; Department of Ophthalmology, Tufts University of Medicine, Boston, MA 02111, USA; The Howard Hughes…

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Molecular therapy for Glaucoma: Astaxanthin, isolated from Haematococcus pluvialis, arctic shrimp among others, increases Nmnat1 expression, increases NAD+ levels, improves neuronal dysfunction, and protects against glaucoma via down regulation of its target gene, 10/March/2018, 5.55 am

Molecular therapy for Glaucoma: Astaxanthin, isolated from Haematococcus pluvialis, arctic shrimp among others, increases Nmnat1 expression, increases NAD+ levels, improves neuronal dysfunction, and protects against glaucoma via down regulation of its target gene, 10/March/2018, 5.55 am 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A study from the The Jackson Laboratory, Bar Harbor, ME 04609, USA; Department of Ophthalmology, Tufts University of Medicine, Boston, MA 02111, USA; The Howard Hughes…

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Natural product therapy for Glaucoma: Formononetin, isolated from Trifolium Pratense, Maackia Amurensis/Amur Maackia, among others, increases Nmnat1 expression, increases NAD+ levels, improves neuronal dysfunction, and protects against glaucoma via down regulation of its target gene, 27/November/2017, 8.07 am

Natural product therapy for Glaucoma: Formononetin, isolated from Trifolium Pratense, Maackia Amurensis/Amur Maackia, among others, increases Nmnat1 expression, increases NAD+ levels, improves neuronal dysfunction, and protects against glaucoma via down regulation of its target gene, 27/November/2017, 8.07 am 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A study from the The Jackson Laboratory, Bar Harbor, ME 04609, USA; Department of Ophthalmology, Tufts University of Medicine, Boston, MA 02111, USA; The Howard Hughes…

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 Molecular therapy for Glaucoma:  Rosiglitaone, an  insulin sensitizer, increases Nmnat1 expression, increases NAD+ levels, improves neuroanl dysfunction, and protects against glaucoma via down regulation of its target gene, 18/October/2017, 11.25 pm

 Molecular therapy for Glaucoma:  Rosiglitaone, an  insulin sensitizer, increases Nmnat1 expression, increases NAD+ levels, improves neuroanl dysfunction, and protects against glaucoma via down regulation of its target gene, 18/October/2017, 11.25 pm 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A study from the The Jackson Laboratory, Bar Harbor, ME 04609, USA; Department of Ophthalmology, Tufts University of Medicine, Boston, MA 02111, USA; The Howard Hughes…

read more