Heart attack

August 16, 2020

Salidorside-based regenerative cardiovascular therapy: Molecular mechanistic insight into how Salidorside protects against and promotes functional recovery after Myocardial Infarction: Salidorside inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration and survival after myocardial infarction, via up-regulation of its target gene, 16/August/2020, 4.03 pm

Salidorside-based regenerative cardiovascular therapy: Molecular mechanistic insight into how Salidorside protects against and promotes functional recovery after Myocardial Infarction: Salidorside inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration and survival after myocardial infarction, via up-regulation of its target gene, 16/August/2020, 4.03 pm https://genomediscovery.org/wp-content/uploads/2020/08/Salidroside-prevents-cardiomyocyte-dysfunction-and-cardiac-ageing-through-up-regulation-of-PNUTS2.jpg 960 720 https://genomediscovery.org/wp-content/uploads/2020/08/Salidroside-prevents-cardiomyocyte-dysfunction-and-cardiac-ageing-through-up-regulation-of-PNUTS2.jpg August 16, 2020 August 16, 2020

What they say: A recent study from the Institute for Cardiovascular Regeneration, Centre of Molecular Medicine, Frankfurt, Germany shows that “MicroRNA-34a regulates cardiac ageing and function.” This study was published, in the 7 March 2013 issue of of the journal Nature, by Profs. Stefanie…

August 1, 2020

Capillarisin-based regenerative cardiovascular therapy: Molecular mechanistic insight into how Capillarisin protects against and promotes functional recovery after Myocardial Infarction: Capillarisin inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration and survival after myocardial infarction, via up-regulation of its target gene, 1/August/2020, 6.59 am

Capillarisin-based regenerative cardiovascular therapy: Molecular mechanistic insight into how Capillarisin protects against and promotes functional recovery after Myocardial Infarction: Capillarisin inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration and survival after myocardial infarction, via up-regulation of its target gene, 1/August/2020, 6.59 am https://genomediscovery.org/wp-content/uploads/2020/07/Capillarisin-prevents-cardiomyocyte-dysfunction-and-cardiac-ageing-through-up-regulation-of-PNUTS.jpg2_.1.jpg 960 720 https://genomediscovery.org/wp-content/uploads/2020/07/Capillarisin-prevents-cardiomyocyte-dysfunction-and-cardiac-ageing-through-up-regulation-of-PNUTS.jpg2_.1.jpg August 1, 2020 August 1, 2020

What they say: A recent study from the Institute for Cardiovascular Regeneration, Centre of Molecular Medicine, Frankfurt, Germany shows that “MicroRNA-34a regulates cardiac ageing and function.” This study was published, in the 7 March 2013 issue of of the journal Nature, by Profs. Stefanie…

July 28, 2020

Cafestol-based regenerative cardiovascular therapy: Molecular mechanistic insight into how Cafestol protects against and promotes functional recovery after Myocardial Infarction: Cafestol inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration and survival after myocardial infarction, via up-regulation of its target gene, 28/July/2020, 11.05 pm

Cafestol-based regenerative cardiovascular therapy: Molecular mechanistic insight into how Cafestol protects against and promotes functional recovery after Myocardial Infarction: Cafestol inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration and survival after myocardial infarction, via up-regulation of its target gene, 28/July/2020, 11.05 pm https://genomediscovery.org/wp-content/uploads/2020/07/Cafestol-prevents-cardiomyocyte-dysfunction-and-cardiac-ageing-through-up-regulation-of-PNUTS.jpg4_.jpg 960 720 https://genomediscovery.org/wp-content/uploads/2020/07/Cafestol-prevents-cardiomyocyte-dysfunction-and-cardiac-ageing-through-up-regulation-of-PNUTS.jpg4_.jpg July 28, 2020 July 28, 2020

What they say: A recent study from the Institute for Cardiovascular Regeneration, Centre of Molecular Medicine, Frankfurt, Germany shows that “MicroRNA-34a regulates cardiac ageing and function.” This study was published, in the 7 March 2013 issue of of the journal Nature, by Profs. Stefanie…

July 27, 2020

Tanshinone IIA-based regenerative cardiovascular therapy: Molecular mechanistic insight into how Tanshinone IIA protects against and promotes functional recovery after Myocardial Infarction: Tanshinone IIA, inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration and survival after myocardial infarction, via up-regulation of its target gene, 27/July/2020, 7.40 pm

Tanshinone IIA-based regenerative cardiovascular therapy: Molecular mechanistic insight into how Tanshinone IIA protects against and promotes functional recovery after Myocardial Infarction: Tanshinone IIA, inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration and survival after myocardial infarction, via up-regulation of its target gene, 27/July/2020, 7.40 pm https://genomediscovery.org/wp-content/uploads/2020/07/Tanshinone-IIA-prevents-cardiomyocyte-dysfunction-and-cardiac-ageing-through-up-regulation-of-PNUTS.jpg3_.jpg 960 720 https://genomediscovery.org/wp-content/uploads/2020/07/Tanshinone-IIA-prevents-cardiomyocyte-dysfunction-and-cardiac-ageing-through-up-regulation-of-PNUTS.jpg3_.jpg July 27, 2020 July 27, 2020

What they say: A recent study from the Institute for Cardiovascular Regeneration, Centre of Molecular Medicine, Frankfurt, Germany shows that “MicroRNA-34a regulates cardiac ageing and function.” This study was published, in the 7 March 2013 issue of of the journal Nature, by Profs. Stefanie…

December 24, 2019

Ascorbic acid-based regenerative cardiovascular therapy: Molecular mechanistic insight into how Vitamin C/Ascorbic acid protects against and promotes functional recovery after Myocardial Infarction: Vitamin C/Ascorbic acid(AA) inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration and survival after myocardial infarction, via up-regulation of its target gene, 24/December/2019, 11.55 pm

Ascorbic acid-based regenerative cardiovascular therapy: Molecular mechanistic insight into how Vitamin C/Ascorbic acid protects against and promotes functional recovery after Myocardial Infarction: Vitamin C/Ascorbic acid(AA) inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration and survival after myocardial infarction, via up-regulation of its target gene, 24/December/2019, 11.55 pm https://genomediscovery.org/wp-content/uploads/2019/12/Ascorbic-acid-or-Vitamin-C-attenuates-cardiomyocyte-apoptosis-and-cardiac-ageing-through-up-regulation-of-PNUTS.jpg 1280 720 https://genomediscovery.org/wp-content/uploads/2019/12/Ascorbic-acid-or-Vitamin-C-attenuates-cardiomyocyte-apoptosis-and-cardiac-ageing-through-up-regulation-of-PNUTS.jpg December 24, 2019 December 29, 2019

What they say: A recent study from the Institute for Cardiovascular Regeneration, Centre of Molecular Medicine, Frankfurt, Germany shows that “MicroRNA-34a regulates cardiac ageing and function.” This study was published, in the 7 March 2013 issue of of the journal Nature, by Profs. Stefanie…

October 17, 2019

Molecular mechanistic insights into how Prasugrel attenuates pathogenesis-associated with Myocardial infarction and protects against myocardial dysfunction: Prasugrel (Trade name: Effient, Efient), a drug used to prevent blood clots, decreases IRF3, GM-CSF (Granulocyte-macrophage colony-stimulating factor) and GRK2(G protein-coupled receptor kinase) expression, inhibits undue leukocyte activation and invasion, suppresses recruitment of inflammatory cells, inhibits ventricular dilation, and promotes heart repair and survival, via up regulation of its target genes, 17/October/2019, 11.29 pm

Molecular mechanistic insights into how Prasugrel attenuates pathogenesis-associated with Myocardial infarction and protects against myocardial dysfunction: Prasugrel (Trade name: Effient, Efient), a drug used to prevent blood clots, decreases IRF3, GM-CSF (Granulocyte-macrophage colony-stimulating factor) and GRK2(G protein-coupled receptor kinase) expression, inhibits undue leukocyte activation and invasion, suppresses recruitment of inflammatory cells, inhibits ventricular dilation, and promotes heart repair and survival, via up regulation of its target genes, 17/October/2019, 11.29 pm https://genomediscovery.org/wp-content/uploads/2019/10/Prasugrel-attenuates-pathogenesis-associated-with-myocardial-infarction4.jpg 960 720 https://genomediscovery.org/wp-content/uploads/2019/10/Prasugrel-attenuates-pathogenesis-associated-with-myocardial-infarction4.jpg October 17, 2019 October 18, 2019

Introduction: What they say: A recent study from Department of Systems Biology, Harvard Medical School, Boston, Massachusetts, USA; and Cardiovascular Research Center, Massachusetts General Hospital and Harvard Medical School, Boston,…

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