Ideas

August 13, 2020

Rare sugar-based therapy for COVID-19 and other infectious diseases: D-Allose-based therapy for SARS-COV-2: D-Allose, the C-3 epimer of glucose, inhibits SARS-COV-2, Hepatitis-B/C, Dengue, Zika, Ebola, HIV-1, Mtb, Malaria, CMV, Influenza H1NI1, Foot and Mouth disease, ADV, respiratory syncytial, Sindbis, and SFV viruses and others by increasing the levels of IFN (Interferon)-alpah/alpha-2/beta/gamma/lamda, interferon-inducible transmembrane protein IFITM3, Interferon-stimulated gene-15, Mx2 and antimicrobial peptides 13/August/2020, 2.55 pm

Rare sugar-based therapy for COVID-19 and other infectious diseases: D-Allose-based therapy for SARS-COV-2: D-Allose, the C-3 epimer of glucose, inhibits SARS-COV-2, Hepatitis-B/C, Dengue, Zika, Ebola, HIV-1, Mtb, Malaria, CMV, Influenza H1NI1, Foot and Mouth disease, ADV, respiratory syncytial, Sindbis, and SFV viruses and others by increasing the levels of IFN (Interferon)-alpah/alpha-2/beta/gamma/lamda, interferon-inducible transmembrane protein IFITM3, Interferon-stimulated gene-15, Mx2 and antimicrobial peptides 13/August/2020, 2.55 pm https://genomediscovery.org/wp-content/uploads/2020/08/D-Allose-attenautes-the-production-of-COVID-19-SARS-COV-2-1.jpg 1280 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2020/08/D-Allose-attenautes-the-production-of-COVID-19-SARS-COV-2-1.jpg August 13, 2020 August 13, 2020

Introduction: What they say: A study from the Ragon Institute of Massachusetts General Hospital, Massachusetts Institute of Technology, and Harvard Medical School, Charlestown, MA 02129, USA shows that “The IFITM proteins…

August 1, 2020

Natural product-based therapy for COVID-19 and other infectious diseases: Fucoidan-based therapy for SARS-COV-2: Fucoidan, a fucose-enriched and sulfated polysaccharide, inhibits SARS-COV-2/1, Hepatitis-B/C, Dengue, Zika, Ebola, HIV-1, Mtb, Malaria, CMV, Influenza H1NI1, Foot and Mouth disease, ADV, respiratory syncytial, Sindbis, and SFV viruses by increasing the Levels of the Antiviral Proteins IFITM3, Interferon-stimulated gene 15, and others 1/August/2020, 9.08 am

Natural product-based therapy for COVID-19 and other infectious diseases: Fucoidan-based therapy for SARS-COV-2: Fucoidan, a fucose-enriched and sulfated polysaccharide, inhibits SARS-COV-2/1, Hepatitis-B/C, Dengue, Zika, Ebola, HIV-1, Mtb, Malaria, CMV, Influenza H1NI1, Foot and Mouth disease, ADV, respiratory syncytial, Sindbis, and SFV viruses by increasing the Levels of the Antiviral Proteins IFITM3, Interferon-stimulated gene 15, and others 1/August/2020, 9.08 am https://genomediscovery.org/wp-content/uploads/2020/08/Oligo-Fucoidan-aids-in-attenuation-and-prevention-of-COVID-19-SARS-COV-21-1.jpg 1280 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2020/08/Oligo-Fucoidan-aids-in-attenuation-and-prevention-of-COVID-19-SARS-COV-21-1.jpg August 1, 2020 August 1, 2020

August 1, 2020

Capillarisin-based regenerative cardiovascular therapy: Molecular mechanistic insight into how Capillarisin protects against and promotes functional recovery after Myocardial Infarction: Capillarisin inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration and survival after myocardial infarction, via up-regulation of its target gene, 1/August/2020, 6.59 am

Capillarisin-based regenerative cardiovascular therapy: Molecular mechanistic insight into how Capillarisin protects against and promotes functional recovery after Myocardial Infarction: Capillarisin inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration and survival after myocardial infarction, via up-regulation of its target gene, 1/August/2020, 6.59 am https://genomediscovery.org/wp-content/uploads/2020/07/Capillarisin-prevents-cardiomyocyte-dysfunction-and-cardiac-ageing-through-up-regulation-of-PNUTS.jpg2_.1.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2020/07/Capillarisin-prevents-cardiomyocyte-dysfunction-and-cardiac-ageing-through-up-regulation-of-PNUTS.jpg2_.1.jpg August 1, 2020 August 1, 2020

What they say: A recent study from the Institute for Cardiovascular Regeneration, Centre of Molecular Medicine, Frankfurt, Germany shows that “MicroRNA-34a regulates cardiac ageing and function.” This study was published, in the 7 March 2013 issue of of the journal Nature, by Profs. Stefanie…

July 28, 2020

Cafestol-based regenerative cardiovascular therapy: Molecular mechanistic insight into how Cafestol protects against and promotes functional recovery after Myocardial Infarction: Cafestol inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration and survival after myocardial infarction, via up-regulation of its target gene, 28/July/2020, 11.05 pm

Cafestol-based regenerative cardiovascular therapy: Molecular mechanistic insight into how Cafestol protects against and promotes functional recovery after Myocardial Infarction: Cafestol inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration and survival after myocardial infarction, via up-regulation of its target gene, 28/July/2020, 11.05 pm https://genomediscovery.org/wp-content/uploads/2020/07/Cafestol-prevents-cardiomyocyte-dysfunction-and-cardiac-ageing-through-up-regulation-of-PNUTS.jpg4_.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2020/07/Cafestol-prevents-cardiomyocyte-dysfunction-and-cardiac-ageing-through-up-regulation-of-PNUTS.jpg4_.jpg July 28, 2020 July 28, 2020

July 27, 2020

Tanshinone IIA-based regenerative cardiovascular therapy: Molecular mechanistic insight into how Tanshinone IIA protects against and promotes functional recovery after Myocardial Infarction: Tanshinone IIA, inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration and survival after myocardial infarction, via up-regulation of its target gene, 27/July/2020, 7.40 pm

Tanshinone IIA-based regenerative cardiovascular therapy: Molecular mechanistic insight into how Tanshinone IIA protects against and promotes functional recovery after Myocardial Infarction: Tanshinone IIA, inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration and survival after myocardial infarction, via up-regulation of its target gene, 27/July/2020, 7.40 pm https://genomediscovery.org/wp-content/uploads/2020/07/Tanshinone-IIA-prevents-cardiomyocyte-dysfunction-and-cardiac-ageing-through-up-regulation-of-PNUTS.jpg3_.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2020/07/Tanshinone-IIA-prevents-cardiomyocyte-dysfunction-and-cardiac-ageing-through-up-regulation-of-PNUTS.jpg3_.jpg July 27, 2020 July 27, 2020

July 26, 2020

Why pollution is bad for your heart: Mechanistic and functional insights into how Pollutant Particular matter (PM2.5) accelerates cardiac dysfunction: Particulate matter (PM2.5) augments DNA damage responses, attenuates telomerase expression, promotes telomere shortening, and attenuates cardiomyocyte survival after myocardial infarction, via down regulation of PNUTS and others, 26/July/2020, 10.45 am

Why pollution is bad for your heart: Mechanistic and functional insights into how Pollutant Particular matter (PM2.5) accelerates cardiac dysfunction: Particulate matter (PM2.5) augments DNA damage responses, attenuates telomerase expression, promotes telomere shortening, and attenuates cardiomyocyte survival after myocardial infarction, via down regulation of PNUTS and others, 26/July/2020, 10.45 am https://genomediscovery.org/wp-content/uploads/2020/07/Particular-matter-PM2.5-prevents-functional-recovery-after-Myocardial-infarction-and-accelerates-cardiac-dysfunction1.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2020/07/Particular-matter-PM2.5-prevents-functional-recovery-after-Myocardial-infarction-and-accelerates-cardiac-dysfunction1.jpg July 26, 2020 July 27, 2020

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