Androgens are called into action to attenuate arthritic pain: Testosterone-based therapy for inflammation and arthritis:Testosterone, a male reproductive hormone, increases IL-9 levels, augments proliferation of ILC2s (type 2 innate lymphoid cells) and regulatory T (Treg) cells, promotes resolution of inflammation, attenuates cartilage destruction, decreases bone loss, and inhibits pathological features associated with arthritis via up-regulation of its target genes, 11/August/2018, 8.25 am
Introduction: What they say A study from Department of Internal Medicine 3-Rheumatology and Immunology, Friedrich Alexander University Erlangen-Nürnberg (FAU) and Universitätsklinikum Erlangen, Erlangen, Germany shows that “Resolution of inflammation by interleukin-9-producing type 2 innate lymphoid cells.” This research paper was published, in the 17 July 2017 issue of the journal “Nature Medicine” [One of the […]
A candy way to say no to pain: Rare sugar-based therapy for inflammation and arthritis:D-Allose, an aldohexose sugar and C-3 epimer of glucose, increases IL-9 levels, augments proliferation of ILC2s (type 2 innate lymphoid cells) and regulatory T (Treg) cells, promotes resolution of inflammation, attenuates cartilage destruction, decreases bone loss, and inhibits pathological features associated with arthritis via up-regulation of its target genes, 18/March/2018, 9.34 am
Introduction: What they say A study from Department of Internal Medicine 3-Rheumatology and Immunology, Friedrich Alexander University Erlangen-Nürnberg (FAU) and Universitätsklinikum Erlangen, Erlangen, Germany shows that “Resolution of inflammation by interleukin-9-producing type 2 innate lymphoid cells.” This research paper was published, in the 17 July 2017 issue of the journal “Nature Medicine” [One of the […]
Natural product-derived therapy for Inflammatory bowel disease: Genistein, isolated from Soy products, promotes NFKB-p50/p65 dimer formation, induces inflammatory gene expression downstream of RelA/p65, inhibits colonic pathogenesis, and stalls the progression of inflammatory bowel disease and colitis via down regulation of its target gene, 24/May/2017, 7.36 am
Introduction: What they say: A study from the Institute for Molecular Medicine, University Medical Center of the Johannes Gutenberg, University of Mainz, Obere Zahlbarer Str, Mainz, Germany shows that increased levels of Bcl-3 inhibits Treg development and function and thereby promotes spontaneous colitis. This study was published, in the 28 April 2017 issue of the […]
Molecular therapy for Inflammatory Bowel disease (IBD): E2F3 preserves the functions of Beclin 1 and ATG5 and decreases tissue injury in inflammatory bowel disease (IBD) via down regulation of its target gene, 08/February/2015, 23.32
A study from Department of Medicine, University of Chicago, Chicago, Illinois, USA shows that “Cytosolic HMGB1 controls the cellular autophagy/apoptosis checkpoint during inflammation.” This study was published in the February 2, 2014 issue of J Clin Invest by Prof. Eugene B. Chang & Dr. Xiaorong Zhu. On the foundation of this interesting finding, Dr L Boominathan, Director-cum-chief Scientist of GBMD, reports here that: Molecular therapy for Inflammatory […]
Therapeutic insights into improving intestinal immunity: Tumor suppressor p53 inhibits the progression of inflammatory bowel disease (IBD) and colitis-associated colorectal cancer via up regulation of its target gene , 18/July/2014, 8.55 am
Idea Proposed/Formulated by: Dr L Boominathan PhD Web: http://genomediscovery.org Terms & Conditions apply http://genomediscovery.org/registration/terms-and-conditions/ Citation: Boominathan,Therapeutic insights into improving intestinal immunity: Tumor suppressor p53 inhibits the progression of inflammatory bowel disease (IBD) and colitis-associated colorectal cancer via up regulation of its target gene , 18/July/2014, 8.55 am, Genome-2-Bio-Medicine Discovery center (GBMD), http://genomediscovery.org Courtesy: When you cite drop us a line at info@genomediscovery.org * Research cooperation
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