Malaria

March 10, 2020

Anti-asthmatic drug comes to the rescue of blood-stage Plasmodium infection: Pranlukast, an anti-asthmatic drug, decreases the expression of CTLA-4, PD-L1 and LAG-3, promotes CD4+ T-cell function, increases secretion of protective antibodies, promotes and clears blood-stage malaria, via up regulation of its target gene, 10/March/2020, 7.38 am

Anti-asthmatic drug comes to the rescue of blood-stage Plasmodium infection: Pranlukast, an anti-asthmatic drug, decreases the expression of CTLA-4, PD-L1 and LAG-3, promotes CD4+ T-cell function, increases secretion of protective antibodies, promotes and clears blood-stage malaria, via up regulation of its target gene, 10/March/2020, 7.38 am https://genomediscovery.org/wp-content/uploads/2020/03/Pranlukast-inhibits-bloodstage-malaria-through-down-regulation-of-immune-evasion-molecules-including-CTLA4-PD-1-PD-L1-and-LAG3.jpg2_.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2020/03/Pranlukast-inhibits-bloodstage-malaria-through-down-regulation-of-immune-evasion-molecules-including-CTLA4-PD-1-PD-L1-and-LAG3.jpg2_.jpg March 10, 2020 March 10, 2020

Introduction: What they say A study from the Department of Microbiology and Immunology, University of Iowa Carver College of Medicine, Iowa City, Iowa, USA shows that “Regulatory T cells impede…

February 28, 2020

Repurposing the anti-hypertensive agent into an anti-malarial agent: Nifidipine, a calcium channel blocker, increases Ferroportin expression, decreases intracellular iron accumulation, inhibits cellular damage, prevents hemolysis, and inhibits malaria infection, via upregulation of its target gene, 28/February/2020, 9.39 am

Repurposing the anti-hypertensive agent into an anti-malarial agent: Nifidipine, a calcium channel blocker, increases Ferroportin expression, decreases intracellular iron accumulation, inhibits cellular damage, prevents hemolysis, and inhibits malaria infection, via upregulation of its target gene, 28/February/2020, 9.39 am https://genomediscovery.org/wp-content/uploads/2020/02/Nifidipine-protects-against-malarial-infection-through-up-regulation-of-ferroportin1-1.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2020/02/Nifidipine-protects-against-malarial-infection-through-up-regulation-of-ferroportin1-1.jpg February 28, 2020 February 28, 2020

National Science day special ********* We wish everyone a very happy national science day. On this special occasion, we are happy to announce that Bio-Med/Pharma/Therapeutic/Clinical ideas posted today (28/February/2020) will be…

February 28, 2019

Probiotic comes to the rescue of blood-stage Plasmodium infection: Probiotic Bifidobacterium adolescentis IM38 decreases the expression of CTLA-4, PD-L1 and LAG-3, promotes CD4+ T-cell function, increases secretion of protective antibodies, promotes and clears blood-stage malaria, via up regulation of its target gene, 28/February/2019, 7.26 am

Probiotic comes to the rescue of blood-stage Plasmodium infection: Probiotic Bifidobacterium adolescentis IM38 decreases the expression of CTLA-4, PD-L1 and LAG-3, promotes CD4+ T-cell function, increases secretion of protective antibodies, promotes and clears blood-stage malaria, via up regulation of its target gene, 28/February/2019, 7.26 am https://genomediscovery.org/wp-content/uploads/2019/02/Probiotic-Bifidobacterium-adolescentis-IM38-inhibits-blood-stage-malaria-through-down-regulation-of-immune-evasion-molecules-including-CTLA4-PD1-PDL1-and-LAG3.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2019/02/Probiotic-Bifidobacterium-adolescentis-IM38-inhibits-blood-stage-malaria-through-down-regulation-of-immune-evasion-molecules-including-CTLA4-PD1-PDL1-and-LAG3.jpg February 28, 2019 February 28, 2019

February 28, 2019

Probiotics-based adjunctive therapy for Malaria: A probiotic mixture(LB+LC) encompassing Lactobacillus plantarum KY1032 and Lactobacillus curvatus HY7601 increases Ferroportin expression, decreases intracellular iron accumulation, inhibits cellular damage, prevents hemolysis, and inhibits malaria infection, via upregulation of its target gene, 28/February/2019, 7.04 am

Probiotics-based adjunctive therapy for Malaria: A probiotic mixture(LB+LC) encompassing Lactobacillus plantarum KY1032 and Lactobacillus curvatus HY7601 increases Ferroportin expression, decreases intracellular iron accumulation, inhibits cellular damage, prevents hemolysis, and inhibits malaria infection, via upregulation of its target gene, 28/February/2019, 7.04 am https://genomediscovery.org/wp-content/uploads/2019/02/A-Probiotic-mixture-consisting-of-Lactobacillus-plantarum-KY1032-Lactobacillus-curvatus-HY7601-protects-against-malarial-infection-through-up-regulation-of-ferroportin.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2019/02/A-Probiotic-mixture-consisting-of-Lactobacillus-plantarum-KY1032-Lactobacillus-curvatus-HY7601-protects-against-malarial-infection-through-up-regulation-of-ferroportin.jpg February 28, 2019 February 28, 2019

February 14, 2019

Molecular therapy for Malaria: Probucol, a drug used in the treatment of dyslipidemia, increases Ferroportin expression, decreases intracellular iron accumulation, inhibits cellular damage, prevents hemolysis, and inhibits malaria infection, via upregulation of its target gene, 14/February/2019, 6.21 am

Molecular therapy for Malaria: Probucol, a drug used in the treatment of dyslipidemia, increases Ferroportin expression, decreases intracellular iron accumulation, inhibits cellular damage, prevents hemolysis, and inhibits malaria infection, via upregulation of its target gene, 14/February/2019, 6.21 am https://genomediscovery.org/wp-content/uploads/2019/02/Probofol-attenuates-malaria-through-up-regulation-of-ferroptin.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2019/02/Probofol-attenuates-malaria-through-up-regulation-of-ferroptin.jpg February 14, 2019 February 14, 2019

Introduction: What they say: A study from the Section on Human Iron Metabolism, Eunice Kennedy Shriver National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, MD…

December 5, 2018

Probiotic-based adjunctive therapy for Malaria: Probiotic Lactobacillus reuteri (DSM 17938 and ATCC PTA 5289) increases Ferroportin expression, decreases intracellular iron accumulation, inhibits cellular damage, prevents hemolysis, and inhibits malaria infection, via upregulation of its target gene, 5/December/2018, 9.35 pm

Probiotic-based adjunctive therapy for Malaria: Probiotic Lactobacillus reuteri (DSM 17938 and ATCC PTA 5289) increases Ferroportin expression, decreases intracellular iron accumulation, inhibits cellular damage, prevents hemolysis, and inhibits malaria infection, via upregulation of its target gene, 5/December/2018, 9.35 pm https://genomediscovery.org/wp-content/uploads/2018/12/Probiotic-Lactobacillus-reuteri-protects-against-malarial-infection-through-up-regulation-of-ferroportin.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2018/12/Probiotic-Lactobacillus-reuteri-protects-against-malarial-infection-through-up-regulation-of-ferroportin.jpg December 5, 2018 December 5, 2018

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