Malaria

January 14, 2018

Natural product-derived therapy for blood-stage Plasmodium infection: Berberine, isolated from Berberis among others, decreases the expression of CTLA-4, PD-L1 and LAG-3, promotes CD4+ T-cell function, increases secretion of protective antibodies, promotes and clear blood-stage malaria via up regulation of its target gene, 15/January/2018, 5.17 am

Natural product-derived therapy for blood-stage Plasmodium infection: Berberine, isolated from Berberis among others, decreases the expression of CTLA-4, PD-L1 and LAG-3, promotes CD4+ T-cell function, increases secretion of protective antibodies, promotes and clear blood-stage malaria via up regulation of its target gene, 15/January/2018, 5.17 am https://genomediscovery.org/wp-content/uploads/2018/01/Berberine-inhibits-blood-stage-malaria-through-down-regulation-of-immune-evasion-molecules-including-CTLA-4-PD-1-PD-L1-and-LAG-3-1.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2018/01/Berberine-inhibits-blood-stage-malaria-through-down-regulation-of-immune-evasion-molecules-including-CTLA-4-PD-1-PD-L1-and-LAG-3-1.jpg January 14, 2018 January 14, 2018

Introduction: What they say A study from the Department of Microbiology and Immunology, University of Iowa Carver College of Medicine, Iowa City, Iowa, USA shows that “Regulatory T cells impede…

January 8, 2018

Molecular therapy for blood-stage Plasmodium infection: Amiloride (trade name Midamor), an anti-hypertensive drug, decreases the expression of CTLA-4, PD-L1 and LAG-3, promotes CD4+ T-cell function, increases secretion of protective antibodies, promotes and clear blood-stage malaria via up regulation of its target gene, 8/January/2017, 10.12 pm

Molecular therapy for blood-stage Plasmodium infection: Amiloride (trade name Midamor), an anti-hypertensive drug, decreases the expression of CTLA-4, PD-L1 and LAG-3, promotes CD4+ T-cell function, increases secretion of protective antibodies, promotes and clear blood-stage malaria via up regulation of its target gene, 8/January/2017, 10.12 pm https://genomediscovery.org/wp-content/uploads/2018/01/Amiloride-inhibits-blood-stage-malaria-through-down-regulation-of-immune-evasion-molecules-including-CTLA-4-PD-1-PD-L1-and-LAG-31.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2018/01/Amiloride-inhibits-blood-stage-malaria-through-down-regulation-of-immune-evasion-molecules-including-CTLA-4-PD-1-PD-L1-and-LAG-31.jpg January 8, 2018 January 8, 2018

Introduction: What they say A study from the Department of Microbiology and Immunology, University of Iowa Carver College of Medicine, Iowa City, Iowa, USA shows that “Regulatory T cells impede…

December 21, 2017

Molecular therapy for blood-stage Plasmodium infection: Metformin, the anti-hyperglycemic medication, decreases the expression of CTLA-4, PD-L1 and LAG-3, promotes CD4+ T-cell function, increases secretion of protective antibodies, promotes and clear blood-stage malaria via up regulation of its target gene, 21/December/2017, 6.58 am

Molecular therapy for blood-stage Plasmodium infection: Metformin, the anti-hyperglycemic medication, decreases the expression of CTLA-4, PD-L1 and LAG-3, promotes CD4+ T-cell function, increases secretion of protective antibodies, promotes and clear blood-stage malaria via up regulation of its target gene, 21/December/2017, 6.58 am https://genomediscovery.org/wp-content/uploads/2017/12/Metformin-inhibits-blood-stage-malaria-through-down-regulation-of-immune-evasion-molecules-including-CTLA-4-PD-1-PD-L1-and-LAG-3-1.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2017/12/Metformin-inhibits-blood-stage-malaria-through-down-regulation-of-immune-evasion-molecules-including-CTLA-4-PD-1-PD-L1-and-LAG-3-1.jpg December 21, 2017 December 21, 2017

Introduction: What they say A study from the Department of Microbiology and Immunology, University of Iowa Carver College of Medicine, Iowa City, Iowa, USA shows that “Regulatory T cells impede…

October 21, 2017

Natural product-derived therapy for blood-stage Plasmodium infection: Andrographolide, isolated from Andrographis Paniculata, decreases the expression of CTLA-4, PD-L1 and LAG-3, promotes CD4+ T-cell function, increases secretion of protective antibodies, promotes and clear blood-stage malaria via up regulation of its target gene, 21/October/2017, 11.34 pm

Natural product-derived therapy for blood-stage Plasmodium infection: Andrographolide, isolated from Andrographis Paniculata, decreases the expression of CTLA-4, PD-L1 and LAG-3, promotes CD4+ T-cell function, increases secretion of protective antibodies, promotes and clear blood-stage malaria via up regulation of its target gene, 21/October/2017, 11.34 pm https://genomediscovery.org/wp-content/uploads/2017/10/Andrographolide-inhibits-blood-stage-malaria-through-down-regulation-of-immune-evasion-molecules-including-CTLA-4-PD-1-PD-L1-and-LAG-3.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2017/10/Andrographolide-inhibits-blood-stage-malaria-through-down-regulation-of-immune-evasion-molecules-including-CTLA-4-PD-1-PD-L1-and-LAG-3.jpg October 21, 2017 October 21, 2017

Introduction: What they say A study from the Department of Microbiology and Immunology, University of Iowa Carver College of Medicine, Iowa City, Iowa, USA shows that “Regulatory T cells impede…

October 16, 2017

Natural product-derived combination therapy for blood-stage Plasmodium infection: A therapeutic mix encompassing Magnolol, Atovaquone, and Honokiol [MAH] decreases the expression of CTLA-4, PD-L1 and LAG-3, promotes CD4+ T-cell function, increases secretion of protective antibodies, promotes and clear blood-stage malaria via up regulation of its target gene, 17/October/2017, 12.50 am

Natural product-derived combination therapy for blood-stage Plasmodium infection: A therapeutic mix encompassing Magnolol, Atovaquone, and Honokiol [MAH] decreases the expression of CTLA-4, PD-L1 and LAG-3, promotes CD4+ T-cell function, increases secretion of protective antibodies, promotes and clear blood-stage malaria via up regulation of its target gene, 17/October/2017, 12.50 am https://genomediscovery.org/wp-content/uploads/2017/10/MAH-acts-as-an-anti-parasitic-agent-against-blood-stage-malaria-through-down-regulation-of-CTLA-4-PD-1-PD-L1-and-LAG-32.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2017/10/MAH-acts-as-an-anti-parasitic-agent-against-blood-stage-malaria-through-down-regulation-of-CTLA-4-PD-1-PD-L1-and-LAG-32.jpg October 16, 2017 October 16, 2017

Introduction: What they say A study from the Department of Microbiology and Immunology, University of Iowa Carver College of Medicine, Iowa City, Iowa, USA shows that “Regulatory T cells impede…

October 8, 2017

Natural product-derived combination therapy for blood-stage Plasmodium infection: A therapeutic mix encompassing Clindamycin, Artemisinin, Doxycyclin, and Quinidine [CADQ] decreases the expression of CTLA-4, PD-L1 and LAG-3, promotes CD4+ T-cell function, increases secretion of protective antibodies, promotes and clear blood-stage malaria via up regulation of its target gene, 8/October/2017, 11.55 pm

Natural product-derived combination therapy for blood-stage Plasmodium infection: A therapeutic mix encompassing Clindamycin, Artemisinin, Doxycyclin, and Quinidine [CADQ] decreases the expression of CTLA-4, PD-L1 and LAG-3, promotes CD4+ T-cell function, increases secretion of protective antibodies, promotes and clear blood-stage malaria via up regulation of its target gene, 8/October/2017, 11.55 pm https://genomediscovery.org/wp-content/uploads/2017/10/CADQ-acts-as-an-anti-parasitic-agent-against-blood-stage-malaria-through-down-regulation-of-CTLA-4-PD-1-PD-L1-and-LAG-31.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2017/10/CADQ-acts-as-an-anti-parasitic-agent-against-blood-stage-malaria-through-down-regulation-of-CTLA-4-PD-1-PD-L1-and-LAG-31.jpg October 8, 2017 October 8, 2017

Introduction: What they say A study from the Department of Microbiology and Immunology, University of Iowa Carver College of Medicine, Iowa City, Iowa, USA shows that “Regulatory T cells impede…

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