Autoimmune diabetes

Omega-3 PUFA-based therapy for blood sugar disease (autoimmune diabetes (TIDM)):  ω-PUFAs (Docosahexaenoic acid (DHA) + Eicosapentaenoic acid (EPA) ) increase PD-L1 expression,  augment Tregs function, promote immune tolerance, increase pancreatic β-cell proliferation and regeneration, increase insulin secretion, improve insulin sensitivity, increase energy utilization, and reverse TIDM, via up regulation of its target gene, 7/April/2018,  11.27 pm

Omega-3 PUFA-based therapy for blood sugar disease (autoimmune diabetes (TIDM)):  ω-PUFAs (Docosahexaenoic acid (DHA) + Eicosapentaenoic acid (EPA) ) increase PD-L1 expression,  augment Tregs function, promote immune tolerance, increase pancreatic β-cell proliferation and regeneration, increase insulin secretion, improve insulin sensitivity, increase energy utilization, and reverse TIDM, via up regulation of its target gene, 7/April/2018,  11.27 pm 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A study from the International Center for T1D, Pediatric Clinical Research Center Romeo ed Enrica Invernizzi, “L. Sacco” Department of Biomedical and Clinical Sciences, University of…

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Omega-3 PUFA-based therapy for Diabetes Mellitus: ω-PUFAs (DHA+EPA) augment the expression of FGF19 and FGF1,  attenuate hepatic glucose production, decrease hepatic acetyl CoA content, bring down the levels of plasma ACTH, and corticosterone, augment insulin sensitivity, promote weight loss and alleviate TIDM, via up regulation of its target gene, 7/April/2018, 11.12 pm

Omega-3 PUFA-based therapy for Diabetes Mellitus: ω-PUFAs (DHA+EPA) augment the expression of FGF19 and FGF1,  attenuate hepatic glucose production, decrease hepatic acetyl CoA content, bring down the levels of plasma ACTH, and corticosterone, augment insulin sensitivity, promote weight loss and alleviate TIDM, via up regulation of its target gene, 7/April/2018, 11.12 pm 960 720 Dr Boomi's Genom-2-Discovery Center

 Introduction: What they say  A study from Diabetes and Obesity Center of Excellence, Department of Medicine, University of Washington, Seattle, Washington, USA shows that Central injection of FGF1 induces sustained…

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Gas therapy for blood sugar disease (autoimmune diabetes (TIDM)): The gasotransmitter Hydrogen sulfide (H2S) increases PD-L1 expression,  augments Tregs function, promotes immune tolerance, increases pancreatic β-cell proliferation and regeneration, increases insulin secretion, improves insulin sensitivity, increases energy utilization, and reverses TIDM, via up regulation of its target gene, 6/April/2018,  7.09 am

Gas therapy for blood sugar disease (autoimmune diabetes (TIDM)): The gasotransmitter Hydrogen sulfide (H2S) increases PD-L1 expression,  augments Tregs function, promotes immune tolerance, increases pancreatic β-cell proliferation and regeneration, increases insulin secretion, improves insulin sensitivity, increases energy utilization, and reverses TIDM, via up regulation of its target gene, 6/April/2018,  7.09 am 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A study from the International Center for T1D, Pediatric Clinical Research Center Romeo ed Enrica Invernizzi, “L. Sacco” Department of Biomedical and Clinical Sciences, University of…

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A sweet way to say no to Diabetes Mellitus: D-Mannose, a C-2 epimer of glucose, augments the expression of FGF19 and FGF1,  attenuates hepatic glucose production, decreases hepatic acetyl CoA content, brings down the levels of plasma ACTH, and corticosterone, augments insulin sensitivity, promotes weight loss and alleviates TIDM via up regulation of its target gene, 2/April/2018, 11.30 am

A sweet way to say no to Diabetes Mellitus: D-Mannose, a C-2 epimer of glucose, augments the expression of FGF19 and FGF1,  attenuates hepatic glucose production, decreases hepatic acetyl CoA content, brings down the levels of plasma ACTH, and corticosterone, augments insulin sensitivity, promotes weight loss and alleviates TIDM via up regulation of its target gene, 2/April/2018, 11.30 am 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say  A study from Diabetes and Obesity Center of Excellence, Department of Medicine, University of Washington, Seattle, Washington, USA shows that Central injection of FGF1 induces sustained…

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A candy way to treat blood sugar disease: Sugar-based therapy for autoimmune diabetes (TIDM): Raffinose, a trisaccharide composed of galactose, glucose and fructose, increases PD-L1 expression, increases Tregs function, promotes immune tolerance, increases pancreatic β-cell proliferation and regeneration, increases insulin secretion, improves insulin sensitivity, increases energy utilization, and reverses TIDM, via up regulation of its target gene, 31/March/2018, 12.15 am

A candy way to treat blood sugar disease: Sugar-based therapy for autoimmune diabetes (TIDM): Raffinose, a trisaccharide composed of galactose, glucose and fructose, increases PD-L1 expression, increases Tregs function, promotes immune tolerance, increases pancreatic β-cell proliferation and regeneration, increases insulin secretion, improves insulin sensitivity, increases energy utilization, and reverses TIDM, via up regulation of its target gene, 31/March/2018, 12.15 am 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A study from the International Center for T1D, Pediatric Clinical Research Center Romeo ed Enrica Invernizzi, “L. Sacco” Department of Biomedical and Clinical Sciences, University of…

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Noble gas therapy for blood sugar disease: Xenon gas inhalation therapy for autoimmune diabetes (TIDM): Inhalation of Xenon gas increases PD-L1 expression,  augments Tregs function, promotes immune tolerance, increases pancreatic β-cell proliferation and regeneration, increases insulin secretion, improves insulin sensitivity, increases energy utilization, and reverses TIDM, via up regulation of its target gene, 26/February/2018,  6.53 am

Noble gas therapy for blood sugar disease: Xenon gas inhalation therapy for autoimmune diabetes (TIDM): Inhalation of Xenon gas increases PD-L1 expression,  augments Tregs function, promotes immune tolerance, increases pancreatic β-cell proliferation and regeneration, increases insulin secretion, improves insulin sensitivity, increases energy utilization, and reverses TIDM, via up regulation of its target gene, 26/February/2018,  6.53 am 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A study from the International Center for T1D, Pediatric Clinical Research Center Romeo ed Enrica Invernizzi, “L. Sacco” Department of Biomedical and Clinical Sciences, University of…

read more