Cardiac hypertrophy and fibrosis

November 28, 2018

Hot-water immersion therapy may safeguard your heart against cardiac dysfunction: Hot-water immersion therapy ( at 39°C for 1 hour) for cardiomyocyte proliferation and heart regeneration: Hot-water immersion therapy decreases tumor suppressor Mir-128 and cyclin-dependent kinase inhibitor p27 expression, increases SUZ12 expression, increases Cyclin E and CDK2 expression, promotes proliferation/re-entry of postnatal/adult cardiomyocytes, attenuates fibrosis, ameliorates cardiac dysfunction, and promotes heart repair in response to myocardial infarction, via up-regulation of its target gene, 28/September/2018, 12.13 am

Hot-water immersion therapy may safeguard your heart against cardiac dysfunction: Hot-water immersion therapy ( at 39°C for 1 hour) for cardiomyocyte proliferation and heart regeneration: Hot-water immersion therapy decreases tumor suppressor Mir-128 and cyclin-dependent kinase inhibitor p27 expression, increases SUZ12 expression, increases Cyclin E and CDK2 expression, promotes proliferation/re-entry of postnatal/adult cardiomyocytes, attenuates fibrosis, ameliorates cardiac dysfunction, and promotes heart repair in response to myocardial infarction, via up-regulation of its target gene, 28/September/2018, 12.13 am https://genomediscovery.org/wp-content/uploads/2018/11/Hot-water-immersion-therapy-attenuates-pathogenesis-associated-with-cardiac-hypertrophy-and-fibrosis.jpg7_.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2018/11/Hot-water-immersion-therapy-attenuates-pathogenesis-associated-with-cardiac-hypertrophy-and-fibrosis.jpg7_.jpg November 28, 2018 December 17, 2019

Introduction: What they say: A recent study from Department of Pathology and Laboratory Medicine, University of Cincinnati College of Medicine, Cincinnati, OH, 45267, USA; Key Laboratory of Molecular Target and…

November 27, 2018

Hot water immersion-based adjuvant therapy for cardiac hypertrophy and fibrosis: Hot water immersion (at 39°C for 1 hour) decreases MiR-29 expression, activates wnt- signaling and its components GSK3B, ICAT/CTNNBIP1, HBP1 and GLIS2, attenuates pathologic hypertrophy, inhibits fibrosis of the heart tissue, and improves cardiac function via upregulation of its target gene, 27/November/2017, 6.48 am

Hot water immersion-based adjuvant therapy for cardiac hypertrophy and fibrosis: Hot water immersion (at 39°C for 1 hour) decreases MiR-29 expression, activates wnt- signaling and its components GSK3B, ICAT/CTNNBIP1, HBP1 and GLIS2, attenuates pathologic hypertrophy, inhibits fibrosis of the heart tissue, and improves cardiac function via upregulation of its target gene, 27/November/2017, 6.48 am https://genomediscovery.org/wp-content/uploads/2018/11/Hot-water-immersion-therapy-attenuates-pathogenesis-associated-with-cardiac-hypertrophy-and-fibrosis.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2018/11/Hot-water-immersion-therapy-attenuates-pathogenesis-associated-with-cardiac-hypertrophy-and-fibrosis.jpg November 27, 2018 November 27, 2018

Introduction: What they say: A recent study from the Institute of Pharmacology and Toxicology, Technical University Munich (TUM), 80802, Munich, Germany; DZHK (German Center for Cardiovascular Research), partner site Munich…

November 15, 2018

Molecular therapy for cardiac dysfunction: Empagliflozin (trade name: Jardiance), used in the treatment of TIIDM, decreases Mir-29 expression, activates Wnt- signaling and its components GSK3B, ICAT/CTNNBIP1, HBP1, and GLIS2, attenuates pathologic hypertrophy, inhibits fibrosis of the heart tissue, and improves cardiac function, via upregulation of its target gene, 15/November/2018, 11.22 pm

Molecular therapy for cardiac dysfunction: Empagliflozin (trade name: Jardiance), used in the treatment of TIIDM, decreases Mir-29 expression, activates Wnt- signaling and its components GSK3B, ICAT/CTNNBIP1, HBP1, and GLIS2, attenuates pathologic hypertrophy, inhibits fibrosis of the heart tissue, and improves cardiac function, via upregulation of its target gene, 15/November/2018, 11.22 pm https://genomediscovery.org/wp-content/uploads/2018/11/Empagliflozin-attenuates-pathogenesis-associated-with-cardiac-hypertrophy-and-fibrosis-through-down-regulation-of-MiR29.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2018/11/Empagliflozin-attenuates-pathogenesis-associated-with-cardiac-hypertrophy-and-fibrosis-through-down-regulation-of-MiR29.jpg November 15, 2018 November 15, 2018

October 28, 2018

Melodious music functions as a protector of your myocardium: Music increases Agrin expression, activates Yap and ERK-mediated signaling, replaces scar tissue with functional cardiomyocytes, and promotes cardiomyocyte regeneration and repair after myocardial infarction, via up-regulation of its target gene, 28/December/2017, 2.07 pm

Melodious music functions as a protector of your myocardium: Music increases Agrin expression, activates Yap and ERK-mediated signaling, replaces scar tissue with functional cardiomyocytes, and promotes cardiomyocyte regeneration and repair after myocardial infarction, via up-regulation of its target gene, 28/December/2017, 2.07 pm https://genomediscovery.org/wp-content/uploads/2018/10/Music-protects-against-diabetic-cardiomyopathy-and-myocardial-infarction.jpg2_.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2018/10/Music-protects-against-diabetic-cardiomyopathy-and-myocardial-infarction.jpg2_.jpg October 28, 2018 October 28, 2018

Introduction: What they say: A recent study from Department of Molecular Cell Biology, Weizmann Institute of Science, Rehovot 76100, Israel; Department of Biomedical Engineering, Duke University, Durham, North Carolina 27708,…

October 18, 2018

The tiny probiotic may safeguard your heart against cardiac dysfunction: Probiotic Lactobacillus pentosus var.plantarum C29-based therapy for cardiomyocyte proliferation and heart regeneration: Probiotic Lactobacillus pentosus var.plantarum C29 decreases tumor suppressor Mir-128 and cyclin-dependent kinase inhibitor p27 expression, increases SUZ12 expression, increases Cyclin E and CDK2 expression, promotes proliferation/re-entry of postnatal/adult cardiomyocytes, attenuates fibrosis, ameliorates cardiac dysfunction, and promotes heart repair in response to myocardial infarction, via up-regulation of its target gene, 18/Ocotober/2018, 11.01 pm

The tiny probiotic may safeguard your heart against cardiac dysfunction: Probiotic Lactobacillus pentosus var.plantarum C29-based therapy for cardiomyocyte proliferation and heart regeneration: Probiotic Lactobacillus pentosus var.plantarum C29 decreases tumor suppressor Mir-128 and cyclin-dependent kinase inhibitor p27 expression, increases SUZ12 expression, increases Cyclin E and CDK2 expression, promotes proliferation/re-entry of postnatal/adult cardiomyocytes, attenuates fibrosis, ameliorates cardiac dysfunction, and promotes heart repair in response to myocardial infarction, via up-regulation of its target gene, 18/Ocotober/2018, 11.01 pm https://genomediscovery.org/wp-content/uploads/2018/10/Probiotic-Lactobacillus-pentosus-var-promotes-cardiac-proliferaiton-and-regeneration.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2018/10/Probiotic-Lactobacillus-pentosus-var-promotes-cardiac-proliferaiton-and-regeneration.jpg October 18, 2018 October 18, 2018

September 28, 2018

Consuming a Probiotic once a day may insulate your heart against cardiac dysfunction: Probiotic Bifidobacterium Breve-based adjunct therapy for cardiac hypertrophy and fibrosis: Probiotic Bifidobacterium Breve decreases Mir-29 expression, activates Wnt- signaling and its components GSK3B, ICAT/CTNNBIP1, HBP1, and GLIS2, attenuates pathologic hypertrophy, inhibits fibrosis of the heart tissue, and improves cardiac function via upregulation of its target gene, 28/September/2018, 3.09 pm

Consuming a Probiotic once a day may insulate your heart against cardiac dysfunction: Probiotic Bifidobacterium Breve-based adjunct therapy for cardiac hypertrophy and fibrosis: Probiotic Bifidobacterium Breve decreases Mir-29 expression, activates Wnt- signaling and its components GSK3B, ICAT/CTNNBIP1, HBP1, and GLIS2, attenuates pathologic hypertrophy, inhibits fibrosis of the heart tissue, and improves cardiac function via upregulation of its target gene, 28/September/2018, 3.09 pm https://genomediscovery.org/wp-content/uploads/2018/09/b.breve-protects-against-cardiac-hypertrophy-and-fibrosis-through-down-regulation-of-miR-29.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2018/09/b.breve-protects-against-cardiac-hypertrophy-and-fibrosis-through-down-regulation-of-miR-29.jpg September 28, 2018 October 20, 2018

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