Diabetes

GDF8-based therapy for hyperglycemia: GDF8 increases the expression of IGF1R, INSR, and IRS2, and promotes an insulin-sensitized state, via up-regulation of  its target gene Lin28, 2/May/2019, 1.47 pm
GDF8-based therapy for hyperglycemia: GDF8 increases the expression of IGF1R, INSR, and IRS2, and promotes an insulin-sensitized state, via up-regulation of  its target gene Lin28, 2/May/2019, 1.47 pm 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say:   A study from the Stem Cell Transplantation Program, Division of Pediatric Hematology/Oncology, Boston Children’s Hospital and Dana-Farber Cancer Institute, Boston, Massachusetts, USA shows that “The Lin28/let-7 axis regulates glucose metabolism.” This study…

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A sweet mix to rescue Diabetes-too good to be true: Isomaltulose (IM)/Trehalose (TH) [IMTH]-based regenerative therapy for reversing diabetes: A pharmaceutical mixture encompassing Isomaltulose (IM) and Trehalose (TH) [IMHT]  increases the expression of Sox17, Sox2, Pdx-1, and Ngn3, decreases mTOR expression, promotes regeneration of insulin-producing ß cells, increases insulin secretion, promotes glucose homeostasis and reverses T1D and T2D via down regulation of its target gene, 2/May/2018, 1.32 pm
A sweet mix to rescue Diabetes-too good to be true: Isomaltulose (IM)/Trehalose (TH) [IMTH]-based regenerative therapy for reversing diabetes: A pharmaceutical mixture encompassing Isomaltulose (IM) and Trehalose (TH) [IMHT]  increases the expression of Sox17, Sox2, Pdx-1, and Ngn3, decreases mTOR expression, promotes regeneration of insulin-producing ß cells, increases insulin secretion, promotes glucose homeostasis and reverses T1D and T2D via down regulation of its target gene, 2/May/2018, 1.32 pm 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A study from the Longevity Institute, School of Gerontology, Department of Biological Sciences, University of Southern California, 3715 McClintock Avenue, Los Angeles, California, USA; and Koch Institute…

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A sweet way to say no to Diabetes-too good to be true: Palatinose-based regenerative therapy for reversing diabetes: Isomaltulose(also known as Palatinose), an artificial sweetener consisting of  glucose and fructose,  increases the expression of Sox17, Sox2, Pdx-1, and Ngn3, decreases mTOR expression, promotes regeneration of insulin-producing ß cells, increases insulin secretion, promotes glucose homeostasis and reverses T1D and T2D via down regulation of its target gene, 1/May/2018, 12.05 am
A sweet way to say no to Diabetes-too good to be true: Palatinose-based regenerative therapy for reversing diabetes: Isomaltulose(also known as Palatinose), an artificial sweetener consisting of  glucose and fructose,  increases the expression of Sox17, Sox2, Pdx-1, and Ngn3, decreases mTOR expression, promotes regeneration of insulin-producing ß cells, increases insulin secretion, promotes glucose homeostasis and reverses T1D and T2D via down regulation of its target gene, 1/May/2018, 12.05 am 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A study from the Longevity Institute, School of Gerontology, Department of Biological Sciences, University of Southern California, 3715 McClintock Avenue, Los Angeles, California, USA; and Koch Institute…

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Ghrelin-based therapy for body weight control, energy homeostasis and TIIDM: Ghrelin  decreases CADM1 and its downstream target genes that inhibit glucose-induced insulin secretion, improves insulin sensitivity, increases energy utilization, promotes weight loss and protects from diet-induced obesity and TIIDM, via up-regulation of its target gene, 27/April/2019, 3.57pm
Ghrelin-based therapy for body weight control, energy homeostasis and TIIDM: Ghrelin  decreases CADM1 and its downstream target genes that inhibit glucose-induced insulin secretion, improves insulin sensitivity, increases energy utilization, promotes weight loss and protects from diet-induced obesity and TIIDM, via up-regulation of its target gene, 27/April/2019, 3.57pm 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say  A study from the Max Delbrück Center for Molecular Medicine, Berlin, Germany shows that “Regulation of body weight and energy homeostasis by neuronal cell adhesion molecule1.”…

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Molecular therapy for body weight control, energy homeostasis and TIIDM: Piromelatine,  a melatonin receptor agonist, inhibits DNA methyltransferase 3a (Dnmt3a) expression, increases FGF21 expression, augments insulin sensitivity, increases glucose tolerance, and protects from diet-induced obesity and TIIDM, via up-regulation of its target gene, 26/April/2019, 11.19 pm
Molecular therapy for body weight control, energy homeostasis and TIIDM: Piromelatine,  a melatonin receptor agonist, inhibits DNA methyltransferase 3a (Dnmt3a) expression, increases FGF21 expression, augments insulin sensitivity, increases glucose tolerance, and protects from diet-induced obesity and TIIDM, via up-regulation of its target gene, 26/April/2019, 11.19 pm 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say  A study from Nutritional Sciences and Toxicology Department, University of California, Berkeley, Berkeley, United States shows that “Dnmt3a is an epigenetic mediator of adipose insulin resistance.”…

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Cyclin helix B peptide (CHBP)-based therapy for body weight control, energy homeostasis and TIIDM: Cyclin helix B peptide (CHBP),  derived from Erythropoietin (EPO)/Hematopoietin, inhibits DNA methyltransferase 3a (Dnmt3a) expression, increases FGF21 expression, augments insulin sensitivity, increases glucose tolerance, and protects from diet-induced obesity and TIIDM, via up-regulation of its target gene, 26/April/2019, 10.55 pm
Cyclin helix B peptide (CHBP)-based therapy for body weight control, energy homeostasis and TIIDM: Cyclin helix B peptide (CHBP),  derived from Erythropoietin (EPO)/Hematopoietin, inhibits DNA methyltransferase 3a (Dnmt3a) expression, increases FGF21 expression, augments insulin sensitivity, increases glucose tolerance, and protects from diet-induced obesity and TIIDM, via up-regulation of its target gene, 26/April/2019, 10.55 pm 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say  A study from Nutritional Sciences and Toxicology Department, University of California, Berkeley, Berkeley, United States shows that “Dnmt3a is an epigenetic mediator of adipose insulin resistance.”…

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