Heart attack

A probiotic way to stay free of cardiac diseases: Lactobacillus fermentum -based therapy for attenuating pathogenesis-associated with Myocardial infarction: Lactobacillus fermentum NCIMB 5221 decreases IRF3 (Interferon regulatory transcription factor-3), GM-CSF (Granulocyte-macrophage colony-stimulating factor) and GRK2(G protein-coupled receptor kinase) expression, inhibits undue leucocyte activation and invasion, suppresses recruitment of inflammatory cells, inhibits ventricular dilation, and promotes heart repair and survival, via up-regulation of its target gene, 2/December/2018, 11.11 pm
A probiotic way to stay free of cardiac diseases: Lactobacillus fermentum -based therapy for attenuating pathogenesis-associated with Myocardial infarction: Lactobacillus fermentum NCIMB 5221 decreases IRF3 (Interferon regulatory transcription factor-3), GM-CSF (Granulocyte-macrophage colony-stimulating factor) and GRK2(G protein-coupled receptor kinase) expression, inhibits undue leucocyte activation and invasion, suppresses recruitment of inflammatory cells, inhibits ventricular dilation, and promotes heart repair and survival, via up-regulation of its target gene, 2/December/2018, 11.11 pm 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say: A recent study from Department of Systems Biology, Harvard Medical School, Boston, Massachusetts, USA; and Cardiovascular Research Center, Massachusetts General Hospital and Harvard Medical School, Boston,…

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Probiotic-based Regenerative therapy for regaining the lost cardiomyocytes in Myocardial patients:   Probiotic Lactobacillus fermentum ZYL0401 increases the expression of ERBB2/Her2 and promotes dedifferentiation  of cardiomyocytes, via down-regulation of its target gene, 2/December/2018, 10.29 pm
Probiotic-based Regenerative therapy for regaining the lost cardiomyocytes in Myocardial patients:   Probiotic Lactobacillus fermentum ZYL0401 increases the expression of ERBB2/Her2 and promotes dedifferentiation  of cardiomyocytes, via down-regulation of its target gene, 2/December/2018, 10.29 pm 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A recent study from the Department of Biological Regulation, Weizmann Institute of Science, Rehovot 76100, Israel shows that “ERBB2 triggers mammalian heart regeneration by promoting cardiomyocyte dedifferentiation and proliferation.” This study was published, in the 6 April…

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Steroid-based Regenerative therapy for regaining the lost cardiomyocytes in Myocardial patients:  Prostaglandin E2 (PGE2)/ Dinoprostone (Trade name: PGE2, Cervidil, Propess, others) increases the expression of ERBB2/Her2 and promotes dedifferentiation  of cardiomyocytes, via down-regulation of its target gene, 1/December/2018, 7.47 am
Steroid-based Regenerative therapy for regaining the lost cardiomyocytes in Myocardial patients:  Prostaglandin E2 (PGE2)/ Dinoprostone (Trade name: PGE2, Cervidil, Propess, others) increases the expression of ERBB2/Her2 and promotes dedifferentiation  of cardiomyocytes, via down-regulation of its target gene, 1/December/2018, 7.47 am 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A recent study from the Department of Biological Regulation, Weizmann Institute of Science, Rehovot 76100, Israel shows that “ERBB2 triggers mammalian heart regeneration by promoting cardiomyocyte dedifferentiation and proliferation.” This study was published, in the 6 April…

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A probiotic way to keep the cardiac diseases at bay: Probiotic Lactobacillus reuteri-based Regenerative therapy for regaining the lost cardiomyocytes in Myocardial patients:  Probiotic Lactobacillus reuteri increases the expression of ERBB2/Her2 and promotes dedifferentiation  of cardiomyocytes via down-regulation of its target gene, 30/November/2018, 9.58 am
A probiotic way to keep the cardiac diseases at bay: Probiotic Lactobacillus reuteri-based Regenerative therapy for regaining the lost cardiomyocytes in Myocardial patients:  Probiotic Lactobacillus reuteri increases the expression of ERBB2/Her2 and promotes dedifferentiation  of cardiomyocytes via down-regulation of its target gene, 30/November/2018, 9.58 am 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A recent study from the Department of Biological Regulation, Weizmann Institute of Science, Rehovot 76100, Israel shows that “ERBB2 triggers mammalian heart regeneration by promoting cardiomyocyte dedifferentiation and proliferation.” This study was published, in the 6 April…

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Hot-water immersion therapy may safeguard your heart against cardiac dysfunction: Hot-water immersion therapy ( at 39°C for 1 hour) for cardiomyocyte proliferation and heart regeneration: Hot-water immersion therapy decreases tumor suppressor Mir-128 and cyclin-dependent kinase inhibitor p27 expression, increases SUZ12 expression, increases Cyclin E and CDK2 expression, promotes proliferation/re-entry of postnatal/adult cardiomyocytes, attenuates fibrosis, ameliorates cardiac dysfunction, and promotes heart repair in response to myocardial infarction, via up-regulation of its target gene, 28/September/2018, 12.13 am
Hot-water immersion therapy may safeguard your heart against cardiac dysfunction: Hot-water immersion therapy ( at 39°C for 1 hour) for cardiomyocyte proliferation and heart regeneration: Hot-water immersion therapy decreases tumor suppressor Mir-128 and cyclin-dependent kinase inhibitor p27 expression, increases SUZ12 expression, increases Cyclin E and CDK2 expression, promotes proliferation/re-entry of postnatal/adult cardiomyocytes, attenuates fibrosis, ameliorates cardiac dysfunction, and promotes heart repair in response to myocardial infarction, via up-regulation of its target gene, 28/September/2018, 12.13 am 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say: A recent study from Department of Pathology and Laboratory Medicine, University of Cincinnati College of Medicine, Cincinnati, OH, 45267, USA; Key Laboratory of Molecular Target and…

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 Molecular therapy for safeguarding your heart against cardiac dysfunction: Prostaglandin E2(PGE2)/Dinoprostone  (trade name: Cervidil, Propess, and others), the drug used to induce labor and in others,  decreases tumor suppressor Mir-128 and cyclin-dependent kinase inhibitor p27 expression, increases SUZ12 expression, increases Cyclin E and CDK2 expression, promotes proliferation/re-entry of postnatal/adult cardiomyocytes, attenuates fibrosis, ameliorates cardiac dysfunction, and promotes heart repair in response to myocardial infarction, via up-regulation of its target gene, 27/November/2018, 10.10 am
 Molecular therapy for safeguarding your heart against cardiac dysfunction: Prostaglandin E2(PGE2)/Dinoprostone  (trade name: Cervidil, Propess, and others), the drug used to induce labor and in others,  decreases tumor suppressor Mir-128 and cyclin-dependent kinase inhibitor p27 expression, increases SUZ12 expression, increases Cyclin E and CDK2 expression, promotes proliferation/re-entry of postnatal/adult cardiomyocytes, attenuates fibrosis, ameliorates cardiac dysfunction, and promotes heart repair in response to myocardial infarction, via up-regulation of its target gene, 27/November/2018, 10.10 am 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say: A recent study from Department of Pathology and Laboratory Medicine, University of Cincinnati College of Medicine, Cincinnati, OH, 45267, USA; Key Laboratory of Molecular Target and…

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