Heart attack

Managing glaucoma with branched chain amino acids: Branched chain amino acids increase Nmnat1 expression, increase NAD+ levels, improve neuronal dysfunction, and protect against glaucoma via down regulation of its target gene, 31/March/2018,  9.29 pm
Managing glaucoma with branched chain amino acids: Branched chain amino acids increase Nmnat1 expression, increase NAD+ levels, improve neuronal dysfunction, and protect against glaucoma via down regulation of its target gene, 31/March/2018,  9.29 pm 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A study from the The Jackson Laboratory, Bar Harbor, ME 04609, USA; Department of Ophthalmology, Tufts University of Medicine, Boston, MA 02111, USA; The Howard Hughes…

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Managing heart diseases with essential amino acids:  Essential amino acid-based therapy for Cardiomyopathy: A therapeutic mix encompassing essential amino acids (10g) decreases the expression of Sox6, restores the balance between slow- and fast-twitch myofiber proteins and alleviates Cardiomyopathy via up regulation of its target gene, 31/March/2018, 12.01 am
Managing heart diseases with essential amino acids:  Essential amino acid-based therapy for Cardiomyopathy: A therapeutic mix encompassing essential amino acids (10g) decreases the expression of Sox6, restores the balance between slow- and fast-twitch myofiber proteins and alleviates Cardiomyopathy via up regulation of its target gene, 31/March/2018, 12.01 am 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say   A study from the Department of Cardiology, Boston Children’s Hospital, Harvard Medical School, Boston, Massachusetts, USA; and Harvard Stem Cell Institute, Harvard University, Cambridge, Massachusetts, USA shows that “Trbp regulates heart function through microRNA-mediated Sox6 repression.” This…

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A sweet way to protect your heart: Trisaccharide-based therapy for Myocardial Infarction: Raffinose, a trisaccharide composed of galactose, glucose, and fructose, inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte survival after myocardial infarction, via up regulation of PNUTS, 30/March/2018, 10.54 pm
A sweet way to protect your heart: Trisaccharide-based therapy for Myocardial Infarction: Raffinose, a trisaccharide composed of galactose, glucose, and fructose, inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte survival after myocardial infarction, via up regulation of PNUTS, 30/March/2018, 10.54 pm 960 720 Dr Boomi's Genom-2-Discovery Center

What they say:  A recent study from the Institute for Cardiovascular Regeneration, Centre of Molecular Medicine, Frankfurt, Germany shows that “MicroRNA-34a regulates cardiac ageing and function.” This study was published, in the 7 March  2013 issue of of the journal Nature,  by Prof Dimmler,…

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A noble way to keep cardiac diseases at bay: Noble gas xenon-based cardiac rejuvenation therapy: Inhalation of Xenon gas suppresses tumor suppressor INK4a/p16 expression, and inhibits dilated cardiomyopathy via up regulation of its target gene, 29/March/2018, 6.31 am
A noble way to keep cardiac diseases at bay: Noble gas xenon-based cardiac rejuvenation therapy: Inhalation of Xenon gas suppresses tumor suppressor INK4a/p16 expression, and inhibits dilated cardiomyopathy via up regulation of its target gene, 29/March/2018, 6.31 am 960 720 Dr Boomi's Genom-2-Discovery Center

What we say: Dr L Boominathan PhD, Director-cum-chief Scientist of GBMD, reports that: A noble way to keep cardiac diseases at bay: Noble gas xenon-based cardiac rejuvenation therapy: Inhalation of…

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A noble way to say no to cardiac diseases: Noble gas Xenon-based therapy for attenuating pathogenesis-associated with Myocardial infarction: Inhalation of noble gas Xenon decreases IRF3 (Interferon regulatory transcription factor-3), GM-CSF (Granulocyte-macrophage colony-stimulating factor) and GRK2(G protein-coupled receptor kinase) expression, inhibits undue leucocyte activation and invasion, suppresses recruitment of inflammatory cells, inhibits ventricular dilation, and promotes heart repair and survival, via up regulation of its target gene, 29/March/2018, 5.50 am
A noble way to say no to cardiac diseases: Noble gas Xenon-based therapy for attenuating pathogenesis-associated with Myocardial infarction: Inhalation of noble gas Xenon decreases IRF3 (Interferon regulatory transcription factor-3), GM-CSF (Granulocyte-macrophage colony-stimulating factor) and GRK2(G protein-coupled receptor kinase) expression, inhibits undue leucocyte activation and invasion, suppresses recruitment of inflammatory cells, inhibits ventricular dilation, and promotes heart repair and survival, via up regulation of its target gene, 29/March/2018, 5.50 am 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say: A recent study from Department of Systems Biology, Harvard Medical School, Boston, Massachusetts, USA; and Cardiovascular Research Center, Massachusetts General Hospital and Harvard Medical School, Boston,…

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A sweet way to say no to cardiac diseases: Non-caloric Sugar-based therapy for attenuating pathogenesis-associated with Myocardial infarction: D-Allulose/D-Psicose, a zero-calorie sweet sugar, decreases IRF3 (Interferon regulatory transcription factor-3), GM-CSF (Granulocyte-macrophage colony-stimulating factor) and GRK2(G protein-coupled receptor kinase) expression, inhibits undue leucocyte activation and invasion, suppresses recruitment of inflammatory cells, inhibits ventricular dilation, and promotes heart repair and survival, via up regulation of its target gene, 24/March/2018, 11.42 pm
A sweet way to say no to cardiac diseases: Non-caloric Sugar-based therapy for attenuating pathogenesis-associated with Myocardial infarction: D-Allulose/D-Psicose, a zero-calorie sweet sugar, decreases IRF3 (Interferon regulatory transcription factor-3), GM-CSF (Granulocyte-macrophage colony-stimulating factor) and GRK2(G protein-coupled receptor kinase) expression, inhibits undue leucocyte activation and invasion, suppresses recruitment of inflammatory cells, inhibits ventricular dilation, and promotes heart repair and survival, via up regulation of its target gene, 24/March/2018, 11.42 pm 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say: A recent study from Department of Systems Biology, Harvard Medical School, Boston, Massachusetts, USA; and Cardiovascular Research Center, Massachusetts General Hospital and Harvard Medical School, Boston,…

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