Heart attack

A glass of milk a day keeps the cardiac diseases at bay: Milk product-based therapy for attenuating pathogenesis-associated with Myocardial infarction: Rumenic acid, a conjugated linoleic acid found abundantly in cow’s milk, decreases IRF3 (Interferon regulatory transcription factor-3), GM-CSF (Granulocyte-macrophage colony-stimulating factor) and GRK2(G protein-coupled receptor kinase) expression, inhibits undue leucocyte activation and invasion, suppresses recruitment of inflammatory cells, inhibits ventricular dilation, and promotes heart repair and survival, via up regulation of its target gene, 20/March/2018, 11.04 pm
A glass of milk a day keeps the cardiac diseases at bay: Milk product-based therapy for attenuating pathogenesis-associated with Myocardial infarction: Rumenic acid, a conjugated linoleic acid found abundantly in cow’s milk, decreases IRF3 (Interferon regulatory transcription factor-3), GM-CSF (Granulocyte-macrophage colony-stimulating factor) and GRK2(G protein-coupled receptor kinase) expression, inhibits undue leucocyte activation and invasion, suppresses recruitment of inflammatory cells, inhibits ventricular dilation, and promotes heart repair and survival, via up regulation of its target gene, 20/March/2018, 11.04 pm 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say: A recent study from Department of Systems Biology, Harvard Medical School, Boston, Massachusetts, USA; and Cardiovascular Research Center, Massachusetts General Hospital and Harvard Medical School, Boston,…

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Amino acid-based therapy for Myocardial Infarction:  Tryptophan, an essential amino acid and precursor of the neurotransmitter serotonin and the sleep hormone melatonin, inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte survival after myocardial infarction, via up regulation of PNUTS, 16/March/2018, 6.26 am
Amino acid-based therapy for Myocardial Infarction:  Tryptophan, an essential amino acid and precursor of the neurotransmitter serotonin and the sleep hormone melatonin, inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte survival after myocardial infarction, via up regulation of PNUTS, 16/March/2018, 6.26 am 960 720 Dr Boomi's Genom-2-Discovery Center

What they say:  A recent study from the Institute for Cardiovascular Regeneration, Centre of Molecular Medicine, Frankfurt, Germany shows that “MicroRNA-34a regulates cardiac ageing and function.” This study was published, in the 7 March  2013 issue of of the journal Nature,  by Prof Dimmler,…

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Amino acid-based therapy for Myocardial Infarction:  Isoleucine, a branched-chain amino acid, inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte survival after myocardial infarction, via up regulation of PNUTS, 14/March/2018, 11.13 pm
Amino acid-based therapy for Myocardial Infarction:  Isoleucine, a branched-chain amino acid, inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte survival after myocardial infarction, via up regulation of PNUTS, 14/March/2018, 11.13 pm 960 720 Dr Boomi's Genom-2-Discovery Center

What they say:  A recent study from the Institute for Cardiovascular Regeneration, Centre of Molecular Medicine, Frankfurt, Germany shows that “MicroRNA-34a regulates cardiac ageing and function.” This study was published, in the 7 March  2013 issue of of the journal Nature,  by Prof Dimmler,…

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D-Allulose-based therapy for Myocardial Infarction:  D-Allulose (also known as D-Psicose), a fructose isomer and sweetener found in jackfruit, figs, raisins, wheat and others, inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte survival after myocardial infarction, via up regulation of PNUTS, 13/March/2018, 11.18 pm, 
D-Allulose-based therapy for Myocardial Infarction:  D-Allulose (also known as D-Psicose), a fructose isomer and sweetener found in jackfruit, figs, raisins, wheat and others, inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte survival after myocardial infarction, via up regulation of PNUTS, 13/March/2018, 11.18 pm,  960 720 Dr Boomi's Genom-2-Discovery Center

What they say:  A recent study from the Institute for Cardiovascular Regeneration, Centre of Molecular Medicine, Frankfurt, Germany shows that “MicroRNA-34a regulates cardiac ageing and function.” This study was published, in the 7 March  2013 issue of of the journal Nature,  by Prof Dimmler,…

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D-Xylose-based therapy for Myocardial Infarction:  D-Xylose, a diabetic sweetener in food, inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte survival after myocardial infarction via up regulation of PNUTS, 12/March/2018, 11.08 pm
D-Xylose-based therapy for Myocardial Infarction:  D-Xylose, a diabetic sweetener in food, inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte survival after myocardial infarction via up regulation of PNUTS, 12/March/2018, 11.08 pm 960 720 Dr Boomi's Genom-2-Discovery Center

What they say:  A recent study from the Institute for Cardiovascular Regeneration, Centre of Molecular Medicine, Frankfurt, Germany shows that “MicroRNA-34a regulates cardiac ageing and function.” This study was published, in the 7 March  2013 issue of of the journal Nature,  by Prof Dimmler,…

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Molecular therapy for attenuating pathogenesis-associated with Myocardial infarction: Human or cow’s milk decreases IRF3 (Interferon regulatory transcription factor-3), GM-CSF (Granulocyte-macrophage colony-stimulating factor) and GRK2(G protein-coupled receptor kinase) expression, inhibits undue leucocyte activation and invasion, suppresses recruitment of inflammatory cells, inhibits ventricular dilation, and promotes heart repair and survival, via up regulation of its target gene, 06/March/2018, 5.55 pm
Molecular therapy for attenuating pathogenesis-associated with Myocardial infarction: Human or cow’s milk decreases IRF3 (Interferon regulatory transcription factor-3), GM-CSF (Granulocyte-macrophage colony-stimulating factor) and GRK2(G protein-coupled receptor kinase) expression, inhibits undue leucocyte activation and invasion, suppresses recruitment of inflammatory cells, inhibits ventricular dilation, and promotes heart repair and survival, via up regulation of its target gene, 06/March/2018, 5.55 pm 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say: A recent study from Department of Systems Biology, Harvard Medical School, Boston, Massachusetts, USA; and Cardiovascular Research Center, Massachusetts General Hospital and Harvard Medical School, Boston,…

read more
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