Heart attack

Natural product-derived combinatorial therapy for functional recovery after Myocardial Infarction and prevent cardiac failure: A pharmaceutical mixture encompassing Gambogic acid (GA) and (-)-EpiGalloCatechin-3-Gallate (EGCG) [GA+EGCG] inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte survival after myocardial infarction, via up regulation of PNUTS, Telomerase & MiR-199,  8/June/2019, 12.06 am
Natural product-derived combinatorial therapy for functional recovery after Myocardial Infarction and prevent cardiac failure: A pharmaceutical mixture encompassing Gambogic acid (GA) and (-)-EpiGalloCatechin-3-Gallate (EGCG) [GA+EGCG] inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte survival after myocardial infarction, via up regulation of PNUTS, Telomerase & MiR-199,  8/June/2019, 12.06 am 960 720 Dr Boomi's Genom-2-Discovery Center

What they say:  A recent study from the Institute for Cardiovascular Regeneration, Centre of Molecular Medicine, Frankfurt, Germany shows that “MicroRNA-34a regulates cardiac ageing and function.” This study was published, in the 7 March  2013 issue of Nature,  by Prof Dimmler, Boon, and others. What we…

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Why Chronic stress-induced depression is not good for your heart: Mechanistic insights into chronic stress-induced depression may accelerate cardiac dysfunction: Chronic stress-induced depression augments DNA damage responses, attenuates telomerase expression, promotes telomere shortening, and attenuates cardiomyocyte survival after myocardial infarction, via down regulation of PNUTS, 7/June/2018, 11.01 pm
Why Chronic stress-induced depression is not good for your heart: Mechanistic insights into chronic stress-induced depression may accelerate cardiac dysfunction: Chronic stress-induced depression augments DNA damage responses, attenuates telomerase expression, promotes telomere shortening, and attenuates cardiomyocyte survival after myocardial infarction, via down regulation of PNUTS, 7/June/2018, 11.01 pm 960 720 Dr Boomi's Genom-2-Discovery Center

What they say:  A recent study from the Institute for Cardiovascular Regeneration, Centre of Molecular Medicine, Frankfurt, Germany shows that “MicroRNA-34a regulates cardiac ageing and function.” This study was published, in the 7 March  2013 issue of of the journal Nature,  by Prof Dimmler,…

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Amino acid-based therapy for Myocardial Infarction:  L-Histidine, an essential amino acid and  precursor for Histamine, inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte survival after myocardial infarction, via up regulation of PNUTS, 31/May/2019, 2.49 pm
Amino acid-based therapy for Myocardial Infarction:  L-Histidine, an essential amino acid and  precursor for Histamine, inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte survival after myocardial infarction, via up regulation of PNUTS, 31/May/2019, 2.49 pm 960 720 Dr Boomi's Genom-2-Discovery Center

What they say:  A recent study from the Institute for Cardiovascular Regeneration, Centre of Molecular Medicine, Frankfurt, Germany shows that “MicroRNA-34a regulates cardiac ageing and function.” This study was published, in the 7 March  2013 issue of of the journal Nature,  by Prof Dimmler,…

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Atrasentan-based Regenerative therapy for regaining the lost cardiomyocytes in Myocardial patients: Atrasentan, an endothelin receptor antagonist selective for subtype A (ETA) used to treat cancer, pulmonary hypertension, diabetic kidney diseases and others, increases the expression of ERBB2/Her2 and promotes dedifferentiation  of cardiomyocytes, via up-regulation of its target gene, 20/May/2019, 12.39 am
Atrasentan-based Regenerative therapy for regaining the lost cardiomyocytes in Myocardial patients: Atrasentan, an endothelin receptor antagonist selective for subtype A (ETA) used to treat cancer, pulmonary hypertension, diabetic kidney diseases and others, increases the expression of ERBB2/Her2 and promotes dedifferentiation  of cardiomyocytes, via up-regulation of its target gene, 20/May/2019, 12.39 am 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say A recent study from the Department of Biological Regulation, Weizmann Institute of Science, Rehovot 76100, Israel shows that “ERBB2 triggers mammalian heart regeneration by promoting cardiomyocyte dedifferentiation and proliferation.” This study was published, in the 6 April…

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Molecular therapy for attenuating pathogenesis-associated with Myocardial infarction: Canagliflozin  (Brand name: Invokana, Sulisent, Prominad, others), an anti-hyperglycemic medication used to treat type II diabetes, decreases GM-CSF (Granulocyte-macrophage colony-stimulating factor) expression, inhibits undue leucocyte activation and invasion, suppresses recruitment of inflammatory cells, inhibits left ventricular rupture, and promotes heart healing via up regulation of its target gene, 17/May/2019, 10.25 pm
Molecular therapy for attenuating pathogenesis-associated with Myocardial infarction: Canagliflozin  (Brand name: Invokana, Sulisent, Prominad, others), an anti-hyperglycemic medication used to treat type II diabetes, decreases GM-CSF (Granulocyte-macrophage colony-stimulating factor) expression, inhibits undue leucocyte activation and invasion, suppresses recruitment of inflammatory cells, inhibits left ventricular rupture, and promotes heart healing via up regulation of its target gene, 17/May/2019, 10.25 pm 960 720 Dr Boomi's Genom-2-Discovery Center

Introduction: What they say: A recent study from Center for Systems Biology, Massachusetts General Hospital and Harvard Medical School, Boston, MA, USA; and Center for Systems Biology, Massachusetts General Hospital…

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Intracardiac/epicardial injection of Carvedilol protects against & promotes repair & functional recovery after Myocardial Infarction: Carvedilol (brand name: Coreg) , used to treat high blood pressure, congestive heart failure, left ventricular dysfunction and others, inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte survival after myocardial infarction, via up-regulation of its target gene, 15/May/2019, 11.47 pm
Intracardiac/epicardial injection of Carvedilol protects against & promotes repair & functional recovery after Myocardial Infarction: Carvedilol (brand name: Coreg) , used to treat high blood pressure, congestive heart failure, left ventricular dysfunction and others, inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte survival after myocardial infarction, via up-regulation of its target gene, 15/May/2019, 11.47 pm 960 720 Dr Boomi's Genom-2-Discovery Center

What they say:  A recent study from the Molecular Medicine Laboratory, International Centre for Genetic Engineering and Biotechnology (ICGEB), a United Nations organisation in Trieste, Trieste, Italy, School of Cardiovascular Medicine &…

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