Tuberculosis

October 11, 2018

Probiotic-based therapy for tuberculosis: Probiotic Lactobacillus Brevis deacetylates RelA/p65, inhibits inflammatory gene expression downstream of RelA/p65, inhibits TB pathogenesis, increases the efficacy of anti-TB drug, and promotes M. tuberculosis clearance via upregulation of its target gene, 12/October/2018, 2.45 am

Probiotic-based therapy for tuberculosis: Probiotic Lactobacillus Brevis deacetylates RelA/p65, inhibits inflammatory gene expression downstream of RelA/p65, inhibits TB pathogenesis, increases the efficacy of anti-TB drug, and promotes M. tuberculosis clearance via upregulation of its target gene, 12/October/2018, 2.45 am https://genomediscovery.org/wp-content/uploads/2018/10/Probiotic-Lactobacillus-brevis-protects-against-drug-resistant-Tuberculosis-1.jpg2_-1.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2018/10/Probiotic-Lactobacillus-brevis-protects-against-drug-resistant-Tuberculosis-1.jpg2_-1.jpg October 11, 2018 October 11, 2018

Introduction: What they say: A study from the Singapore Immunology Network, Agency for Science, Technology and Research, Singapore; and Lee Kong Chian School of Medicine, Nanyang Technological University, Singapore shows…

October 10, 2018

Probiotic-based therapy for tuberculosis: Probiotic Lactobacillus gasseri SBT2055 deacetylates RelA/p65, inhibits inflammatory gene expression downstream of RelA/p65, inhibits TB pathogenesis, increases the efficacy of anti-TB drug, and promotes M. tuberculosis clearance via upregulation of its target gene, 11/October/2018, 12.01 am

Probiotic-based therapy for tuberculosis: Probiotic Lactobacillus gasseri SBT2055 deacetylates RelA/p65, inhibits inflammatory gene expression downstream of RelA/p65, inhibits TB pathogenesis, increases the efficacy of anti-TB drug, and promotes M. tuberculosis clearance via upregulation of its target gene, 11/October/2018, 12.01 am https://genomediscovery.org/wp-content/uploads/2018/10/P.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2018/10/P.jpg October 10, 2018 October 10, 2018

April 7, 2018

Steroid-based therapy for tuberculosis: 17-β estradiol, a steroid hormone, deacetylates RelA/p65, inhibits inflammatory gene expression downstream of RelA/p65, inhibits TB pathogenesis, increases the efficacy of anti-TB drug, and promotes M. tuberculosis clearance via up regulation of its target gene, 7/April/2018, 10.54 pm

Steroid-based therapy for tuberculosis: 17-β estradiol, a steroid hormone, deacetylates RelA/p65, inhibits inflammatory gene expression downstream of RelA/p65, inhibits TB pathogenesis, increases the efficacy of anti-TB drug, and promotes M. tuberculosis clearance via up regulation of its target gene, 7/April/2018, 10.54 pm https://genomediscovery.org/wp-content/uploads/2018/04/Estradiol-protects-against-drug-resistant-Tuberculosis-through-deacetylation-of-p65.RelA_.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2018/04/Estradiol-protects-against-drug-resistant-Tuberculosis-through-deacetylation-of-p65.RelA_.jpg April 7, 2018 April 7, 2018

March 18, 2018

Sugar-based therapy for tuberculosis: Lactulose, a non-absorbable sugar used in the treatment of constipation and hepatic encephalopathy, deacetylates RelA/p65, inhibits inflammatory gene expression downstream of RelA/p65, inhibits TB pathogenesis, increases the efficacy of anti-TB drug, and promotes M. tuberculosis clearance via up regulation of its target gene, 18/March/2018, 11.24 am

Sugar-based therapy for tuberculosis: Lactulose, a non-absorbable sugar used in the treatment of constipation and hepatic encephalopathy, deacetylates RelA/p65, inhibits inflammatory gene expression downstream of RelA/p65, inhibits TB pathogenesis, increases the efficacy of anti-TB drug, and promotes M. tuberculosis clearance via up regulation of its target gene, 18/March/2018, 11.24 am https://genomediscovery.org/wp-content/uploads/2018/03/Lactulose-4-O-β-D-Galactosyl-D-fructose-protects-against-drug-resistant-tuberculosis-through-deacetylation-of-p65.jpg2_.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2018/03/Lactulose-4-O-β-D-Galactosyl-D-fructose-protects-against-drug-resistant-tuberculosis-through-deacetylation-of-p65.jpg2_.jpg March 18, 2018 March 18, 2018

March 3, 2018

Molecular therapy for tuberculosis: Carvedilol (brand name: Coreg), used in treatment of congestive heart failure and left ventricular dysfunction, deacetylates RelA/p65, inhibits inflammatory gene expression downstream of RelA/p65, inhibits TB pathogenesis, increases the efficacy of anti-TB drug, and promotes M. tuberculosis clearance via up regulation of its target gene, 3/March/2018, 9.23 am

Molecular therapy for tuberculosis: Carvedilol (brand name: Coreg), used in treatment of congestive heart failure and left ventricular dysfunction, deacetylates RelA/p65, inhibits inflammatory gene expression downstream of RelA/p65, inhibits TB pathogenesis, increases the efficacy of anti-TB drug, and promotes M. tuberculosis clearance via up regulation of its target gene, 3/March/2018, 9.23 am https://genomediscovery.org/wp-content/uploads/2018/03/Carvidilol-attenuates-the-progression-of-drug-resistant-tuberculosis.jpg2_.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2018/03/Carvidilol-attenuates-the-progression-of-drug-resistant-tuberculosis.jpg2_.jpg March 3, 2018 March 3, 2018

March 2, 2018

Molecular therapy for tuberculosis: Triclosan, an antibacterial and antifungal agent found commonly in toothpastes, soaps, among others, deacetylates RelA/p65, inhibits inflammatory gene expression downstream of RelA/p65, inhibits TB pathogenesis, increases the efficacy of anti-TB drug, and promotes M. tuberculosis clearance via up regulation of its target gene, 2/March/2018, 8.06 am

Molecular therapy for tuberculosis: Triclosan, an antibacterial and antifungal agent found commonly in toothpastes, soaps, among others, deacetylates RelA/p65, inhibits inflammatory gene expression downstream of RelA/p65, inhibits TB pathogenesis, increases the efficacy of anti-TB drug, and promotes M. tuberculosis clearance via up regulation of its target gene, 2/March/2018, 8.06 am https://genomediscovery.org/wp-content/uploads/2018/03/Triclosan-protects-against-drug-resistant-Tuberculosis-through-deacetylation-of-p65.RelA_.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2018/03/Triclosan-protects-against-drug-resistant-Tuberculosis-through-deacetylation-of-p65.RelA_.jpg March 2, 2018 March 2, 2018

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