Cardiac regeneration

December 2, 2020

LNCRNA-based regenerative cardiovascular therapy: LncRNA CSMD1-1 inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration and survival after myocardial infarction, via up-regulation of its target gene, 2/December/2020, 9.27 pm

LNCRNA-based regenerative cardiovascular therapy: LncRNA CSMD1-1 inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration and survival after myocardial infarction, via up-regulation of its target gene, 2/December/2020, 9.27 pm https://genomediscovery.org/wp-content/uploads/2020/12/LNCRNA-CSMD1-1-based-therapy-improves-functional-recovery-after-Myocardial-infarction-and-protects-against-myocardial-dysfunction-1.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2020/12/LNCRNA-CSMD1-1-based-therapy-improves-functional-recovery-after-Myocardial-infarction-and-protects-against-myocardial-dysfunction-1.jpg December 2, 2020 December 2, 2020

What they say: A recent study from the Institute for Cardiovascular Regeneration, Centre of Molecular Medicine, Frankfurt, Germany shows that “MicroRNA-34a regulates cardiac ageing and function.” This study was published, in the 7 March 2013 issue of the journal Nature, by Profs. Stefanie Dimmeler…

December 1, 2020

LNCRNA-based regenerative cardiovascular therapy: LncRNA SNGH1 (small nucleolar RNA host gene) inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration, and survival after myocardial infarction, via up-regulation of its target gene, 1/December/2020, 12.55 pm

LNCRNA-based regenerative cardiovascular therapy: LncRNA SNGH1 (small nucleolar RNA host gene) inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration, and survival after myocardial infarction, via up-regulation of its target gene, 1/December/2020, 12.55 pm https://genomediscovery.org/wp-content/uploads/2020/12/Lnc-RNA-SNGH1-prevents-cardiomyocyte-dysfunction-and-cardiac-ageing-through-up-regulation-of-PNUTS-1-1.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2020/12/Lnc-RNA-SNGH1-prevents-cardiomyocyte-dysfunction-and-cardiac-ageing-through-up-regulation-of-PNUTS-1-1.jpg December 1, 2020 December 1, 2020

August 16, 2020

Salidorside-based regenerative cardiovascular therapy: Molecular mechanistic insight into how Salidorside protects against and promotes functional recovery after Myocardial Infarction: Salidorside inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration and survival after myocardial infarction, via up-regulation of its target gene, 16/August/2020, 4.03 pm

Salidorside-based regenerative cardiovascular therapy: Molecular mechanistic insight into how Salidorside protects against and promotes functional recovery after Myocardial Infarction: Salidorside inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration and survival after myocardial infarction, via up-regulation of its target gene, 16/August/2020, 4.03 pm https://genomediscovery.org/wp-content/uploads/2020/08/Salidroside-prevents-cardiomyocyte-dysfunction-and-cardiac-ageing-through-up-regulation-of-PNUTS2.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2020/08/Salidroside-prevents-cardiomyocyte-dysfunction-and-cardiac-ageing-through-up-regulation-of-PNUTS2.jpg August 16, 2020 August 16, 2020

August 1, 2020

Capillarisin-based regenerative cardiovascular therapy: Molecular mechanistic insight into how Capillarisin protects against and promotes functional recovery after Myocardial Infarction: Capillarisin inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration and survival after myocardial infarction, via up-regulation of its target gene, 1/August/2020, 6.59 am

Capillarisin-based regenerative cardiovascular therapy: Molecular mechanistic insight into how Capillarisin protects against and promotes functional recovery after Myocardial Infarction: Capillarisin inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration and survival after myocardial infarction, via up-regulation of its target gene, 1/August/2020, 6.59 am https://genomediscovery.org/wp-content/uploads/2020/07/Capillarisin-prevents-cardiomyocyte-dysfunction-and-cardiac-ageing-through-up-regulation-of-PNUTS.jpg2_.1.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2020/07/Capillarisin-prevents-cardiomyocyte-dysfunction-and-cardiac-ageing-through-up-regulation-of-PNUTS.jpg2_.1.jpg August 1, 2020 August 1, 2020

July 28, 2020

Cafestol-based regenerative cardiovascular therapy: Molecular mechanistic insight into how Cafestol protects against and promotes functional recovery after Myocardial Infarction: Cafestol inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration and survival after myocardial infarction, via up-regulation of its target gene, 28/July/2020, 11.05 pm

Cafestol-based regenerative cardiovascular therapy: Molecular mechanistic insight into how Cafestol protects against and promotes functional recovery after Myocardial Infarction: Cafestol inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration and survival after myocardial infarction, via up-regulation of its target gene, 28/July/2020, 11.05 pm https://genomediscovery.org/wp-content/uploads/2020/07/Cafestol-prevents-cardiomyocyte-dysfunction-and-cardiac-ageing-through-up-regulation-of-PNUTS.jpg4_.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2020/07/Cafestol-prevents-cardiomyocyte-dysfunction-and-cardiac-ageing-through-up-regulation-of-PNUTS.jpg4_.jpg July 28, 2020 July 28, 2020

July 27, 2020

Tanshinone IIA-based regenerative cardiovascular therapy: Molecular mechanistic insight into how Tanshinone IIA protects against and promotes functional recovery after Myocardial Infarction: Tanshinone IIA, inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration and survival after myocardial infarction, via up-regulation of its target gene, 27/July/2020, 7.40 pm

Tanshinone IIA-based regenerative cardiovascular therapy: Molecular mechanistic insight into how Tanshinone IIA protects against and promotes functional recovery after Myocardial Infarction: Tanshinone IIA, inhibits DNA damage responses, induces telomerase expression, inhibits telomere shortening, and promotes cardiomyocyte proliferation, regeneration and survival after myocardial infarction, via up-regulation of its target gene, 27/July/2020, 7.40 pm https://genomediscovery.org/wp-content/uploads/2020/07/Tanshinone-IIA-prevents-cardiomyocyte-dysfunction-and-cardiac-ageing-through-up-regulation-of-PNUTS.jpg3_.jpg 960 720 Dr Boomi's Genom-2-Discovery Center Dr Boomi's Genom-2-Discovery Center https://genomediscovery.org/wp-content/uploads/2020/07/Tanshinone-IIA-prevents-cardiomyocyte-dysfunction-and-cardiac-ageing-through-up-regulation-of-PNUTS.jpg3_.jpg July 27, 2020 July 27, 2020

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