Ideas

Natural product therapy for tuberculosis: γ-Bisabolene, one of the components in Cardamom, Cubeb, lemon, Oregano, among others, increases the expression of MKP-1, inhibits TLR2 signalling, secretion of Chemokine CXCL5, and recruitment of polymorphonuclear leukocytes (PMNs), decreases destructive pulmonary inflammation, promotes M. tuberculosis clearance and inhibits pulmonary tuberculosis via up-regulation of its target gene, 24/October/2016, 10.46 pm
Natural product therapy for tuberculosis: γ-Bisabolene, one of the components in Cardamom, Cubeb, lemon, Oregano, among others, increases the expression of MKP-1, inhibits TLR2 signalling, secretion of Chemokine CXCL5, and recruitment of polymorphonuclear leukocytes (PMNs), decreases destructive pulmonary inflammation, promotes M. tuberculosis clearance and inhibits pulmonary tuberculosis via up-regulation of its target gene, 24/October/2016, 10.46 pm 150 150 Dr Boomi's Genom-2-Discovery Center

A study from the Max Plank Institute for Infection Biology, Berlin, Germany shows that “CXCL5-secreting pulmonary epithelial cells drive destructive neutrophilic inflammation in tuberculosis.” This study was published in the…

read more

Natural product-based therapy for Idiopathic pulmonary fibrosis: Hyperoside, found in Drosera Rotundifolia and Prunella vulgaris (Common self-heal), increases TRF1 expression and ameliorates pulmonary fibrosis via upregulation of its target gene, 24/October/2015, 10.29 pm
Natural product-based therapy for Idiopathic pulmonary fibrosis: Hyperoside, found in Drosera Rotundifolia and Prunella vulgaris (Common self-heal), increases TRF1 expression and ameliorates pulmonary fibrosis via upregulation of its target gene, 24/October/2015, 10.29 pm 150 150 Dr Boomi's Genom-2-Discovery Center

A study from the Spanish National Cancer Centre, Melchor Fernández Almagro 3, Madrid, Spain“ shows that: “Mice with Pulmonary Fibrosis Driven by Telomere Dysfunction.” This study was published in the 14 July 2015 issue of the journal “Cell Reports”…

read more

Molecular therapy for Myocardial Infarction: NGFI-B nuclear orphan receptor Nurr1 improves myocardial function after myocardial infarction via up regulation of PNUTS/PPP1R10 (Serine/threonine-protein phosphatase 1 regulatory subunit 10), 24/October/2016, 10.13 pm
Molecular therapy for Myocardial Infarction: NGFI-B nuclear orphan receptor Nurr1 improves myocardial function after myocardial infarction via up regulation of PNUTS/PPP1R10 (Serine/threonine-protein phosphatase 1 regulatory subunit 10), 24/October/2016, 10.13 pm 150 150 Dr Boomi's Genom-2-Discovery Center

A recent study from the Institute for Cardiovascular Regeneration, Centre of Molecular Medicine, Frankfurt, Germany shows that “MicroRNA-34a regulates cardiac ageing and function.” This study was published in the 7 March  2013 issue of Nature  by Prof Dimmler, Boon, and others. On the foundation of this…

read more

Natural product Lifespan extension therapy: Apple extract increases life span via up-regulation of its target gene BubR1, 24/October/2016, 9.48 pm
Natural product Lifespan extension therapy: Apple extract increases life span via up-regulation of its target gene BubR1, 24/October/2016, 9.48 pm 150 150 Dr Boomi's Genom-2-Discovery Center

A recent study from the Department of Genetics, Paul F. Glenn Laboratories for the Biological Mechanisms of Aging Harvard Medical School, Boston,USA; and Department of Pharmacology, School of Medicine, The University of…

read more

Natural product therapy for Breast Cancer: Hibiscus syriacus (Rose of Sharon) extract increases the levels of tumor suppressor p53, inhibits the proliferation of tumor-initiating cells in breast cancer, suppresses the progression of metastatic breast cancer, and supports tumor relapse-free survival in breast cancer patients, 24/October/2015, 2. 48 pm
Natural product therapy for Breast Cancer: Hibiscus syriacus (Rose of Sharon) extract increases the levels of tumor suppressor p53, inhibits the proliferation of tumor-initiating cells in breast cancer, suppresses the progression of metastatic breast cancer, and supports tumor relapse-free survival in breast cancer patients, 24/October/2015, 2. 48 pm 150 150 Dr Boomi's Genom-2-Discovery Center

A study from the Division of Molecular Medicine and Genetics, Department of Internal Medicine, The Life Sciences Institute, University of Michigan, Ann Arbor, Michigan 48109, USA shows that “Snail1-dependent p53…

read more

Molecular therapy for Myocardial dysfunction: MiR-16 increases CDK inhibitor p27 expression, inhibits mTOR pathway, promotes autophagy in myocardial cells and prevents myocardial dysfunction via down-regulation of its target gene, 24/October/2016, 9.05 am
Molecular therapy for Myocardial dysfunction: MiR-16 increases CDK inhibitor p27 expression, inhibits mTOR pathway, promotes autophagy in myocardial cells and prevents myocardial dysfunction via down-regulation of its target gene, 24/October/2016, 9.05 am 150 150 Dr Boomi's Genom-2-Discovery Center

Dr L Boominathan PhD, Director-cum-chief Scientist of GBMD, reports here that: Molecular therapy for Myocardial dysfunction: MiR-16 increases CDK inhibitor p27 expression, inhibits mTOR pathway, promotes autophagy in myocardial cells and prevents myocardial…

read more
Start Typing